ABSTRACT
Background: Myelomalacia is a multifocal syndrome that causes hemorrhagic infarction of the spinal cord that can occur as a sequel to acute spinal cord injury. It occurs as a consequence of acute spinal cord trauma, usually by extrusion of intervertebral disc in the thoracolumbar spine, affecting up to 10% of dogs with acute paralysis and deep pain loss in the pelvic limbs due to disc disease. This disease may be focal or diffuse; the diffuse form is typically associated with cranial and caudal migration of neurologic signs and is often fatal. The mechanical lesion to the spinal cord triggers secondary injury mediated by the existence of oxygen free radicals, alterations in the concentrations of neurotransmitters ions and amino acids, and it results in the reduction of the spinal cord blood fl ow, ischemia and progressive cranial and caudal necrosis to the focus of the initial injury.Case: A 5-year-old Dachshund male intact dog weighting 8 kg was referred to the Veterinary Teaching Hospital of the Federal University of Rio Grande do Sul (HCV-UFRGS) with initial complaint of acute paralysis of the hind limbs for two days. On clinical examination there was paraplegia, absence of deep pain in the pelvic limbs, with decreased patellar, sciatic and tibial cranial refl exes. The anal sphincter was relaxed and the bladder was easily expressed. The panniculus refl ex was intact up to t
Background: Myelomalacia is a multifocal syndrome that causes hemorrhagic infarction of the spinal cord that can occur as a sequel to acute spinal cord injury. It occurs as a consequence of acute spinal cord trauma, usually by extrusion of intervertebral disc in the thoracolumbar spine, affecting up to 10% of dogs with acute paralysis and deep pain loss in the pelvic limbs due to disc disease. This disease may be focal or diffuse; the diffuse form is typically associated with cranial and caudal migration of neurologic signs and is often fatal. The mechanical lesion to the spinal cord triggers secondary injury mediated by the existence of oxygen free radicals, alterations in the concentrations of neurotransmitters ions and amino acids, and it results in the reduction of the spinal cord blood fl ow, ischemia and progressive cranial and caudal necrosis to the focus of the initial injury.Case: A 5-year-old Dachshund male intact dog weighting 8 kg was referred to the Veterinary Teaching Hospital of the Federal University of Rio Grande do Sul (HCV-UFRGS) with initial complaint of acute paralysis of the hind limbs for two days. On clinical examination there was paraplegia, absence of deep pain in the pelvic limbs, with decreased patellar, sciatic and tibial cranial refl exes. The anal sphincter was relaxed and the bladder was easily expressed. The panniculus refl ex was intact up to t
ABSTRACT
Background: Myelomalacia is a multifocal syndrome that causes hemorrhagic infarction of the spinal cord that can occur as a sequel to acute spinal cord injury. It occurs as a consequence of acute spinal cord trauma, usually by extrusion of intervertebral disc in the thoracolumbar spine, affecting up to 10% of dogs with acute paralysis and deep pain loss in the pelvic limbs due to disc disease. This disease may be focal or diffuse; the diffuse form is typically associated with cranial and caudal migration of neurologic signs and is often fatal. The mechanical lesion to the spinal cord triggers secondary injury mediated by the existence of oxygen free radicals, alterations in the concentrations of neurotransmitters ions and amino acids, and it results in the reduction of the spinal cord blood fl ow, ischemia and progressive cranial and caudal necrosis to the focus of the initial injury.Case: A 5-year-old Dachshund male intact dog weighting 8 kg was referred to the Veterinary Teaching Hospital of the Federal University of Rio Grande do Sul (HCV-UFRGS) with initial complaint of acute paralysis of the hind limbs for two days. On clinical examination there was paraplegia, absence of deep pain in the pelvic limbs, with decreased patellar, sciatic and tibial cranial refl exes. The anal sphincter was relaxed and the bladder was easily expressed. The panniculus refl ex was intact up to t
Background: Myelomalacia is a multifocal syndrome that causes hemorrhagic infarction of the spinal cord that can occur as a sequel to acute spinal cord injury. It occurs as a consequence of acute spinal cord trauma, usually by extrusion of intervertebral disc in the thoracolumbar spine, affecting up to 10% of dogs with acute paralysis and deep pain loss in the pelvic limbs due to disc disease. This disease may be focal or diffuse; the diffuse form is typically associated with cranial and caudal migration of neurologic signs and is often fatal. The mechanical lesion to the spinal cord triggers secondary injury mediated by the existence of oxygen free radicals, alterations in the concentrations of neurotransmitters ions and amino acids, and it results in the reduction of the spinal cord blood fl ow, ischemia and progressive cranial and caudal necrosis to the focus of the initial injury.Case: A 5-year-old Dachshund male intact dog weighting 8 kg was referred to the Veterinary Teaching Hospital of the Federal University of Rio Grande do Sul (HCV-UFRGS) with initial complaint of acute paralysis of the hind limbs for two days. On clinical examination there was paraplegia, absence of deep pain in the pelvic limbs, with decreased patellar, sciatic and tibial cranial refl exes. The anal sphincter was relaxed and the bladder was easily expressed. The panniculus refl ex was intact up to t
ABSTRACT
Introdução: A peritonite consiste em inflamação do peritônio, com classificação variável em relação à origem, ao grau de contaminação e à extensão (localizada ou difusa). A perda da integridade da parede intestinal corresponde à maioria dos casos de peritonite bacteriana nos cães e gatos. Geralmente, a apresentação é aguda em casos de peritonite séptica e crônica na peritonite não séptica. No caso de peritonite séptica, os sinais clínicos constam de desconforto abdominal, letargia, anorexia, perda de peso, vômito, diarréia, distensão abdominal, icterícia variável e colapso. O objetivo deste trabalho é relatar um caso de peritonite séptica secundária à enterotomia em felino.Caso: Um felino, fêmea, castrado, de aproximadamente um ano de idade, foi atendido no Hospital de Clínicas Veterinárias da Universidade Federal do Rio Grande do Sul (HCV-UFRGS), com histórico de ter sido submetida à enterotomia em outro estabelecimento veterinário, devido à formação de fecaloma, uma semana prévia à consulta ao HCV-UFRGS. Os proprietários relataram que alguns dias após o procedimento o animal apresentou-se prostrado, anoréxico e constipado. No exame físico, apresentou dor aguda à palpação abdominal, além de abdômen distendido, dispnéia, mucosas hipocoradas, desidratação e temperatura retal de 39,4C. Os exames de sangue revelaram anemia leve (23%) normocrômica normocítica, leucopenia, hipoprot
ABSTRACT
Introdução: A peritonite consiste em inflamação do peritônio, com classificação variável em relação à origem, ao grau de contaminação e à extensão (localizada ou difusa). A perda da integridade da parede intestinal corresponde à maioria dos casos de peritonite bacteriana nos cães e gatos. Geralmente, a apresentação é aguda em casos de peritonite séptica e crônica na peritonite não séptica. No caso de peritonite séptica, os sinais clínicos constam de desconforto abdominal, letargia, anorexia, perda de peso, vômito, diarréia, distensão abdominal, icterícia variável e colapso. O objetivo deste trabalho é relatar um caso de peritonite séptica secundária à enterotomia em felino.Caso: Um felino, fêmea, castrado, de aproximadamente um ano de idade, foi atendido no Hospital de Clínicas Veterinárias da Universidade Federal do Rio Grande do Sul (HCV-UFRGS), com histórico de ter sido submetida à enterotomia em outro estabelecimento veterinário, devido à formação de fecaloma, uma semana prévia à consulta ao HCV-UFRGS. Os proprietários relataram que alguns dias após o procedimento o animal apresentou-se prostrado, anoréxico e constipado. No exame físico, apresentou dor aguda à palpação abdominal, além de abdômen distendido, dispnéia, mucosas hipocoradas, desidratação e temperatura retal de 39,4C. Os exames de sangue revelaram anemia leve (23%) normocrômica normocítica, leucopenia, hipoprot