ABSTRACT
A 12-year-old spayed female German shorthaired pointer dog sustained extensive bite wounds around the neck. At presentation, atrial fibrillation was identified with a rapid ventricular response rate of 300 beats per minute (bpm). The ventricular response rate rapidly decreased to 130 bpm following administration of hydromorphone and oxygen. Based on the rate, antiarrhythmic therapy was not initiated. The heart rhythm converted back to sinus rhythm by the time of the first recheck evaluation 2 days later, and the dog remained in sinus rhythm at all subsequent evaluations. With the resolution of the arrhythmia, paroxysmal atrial fibrillation was suspected. The underlying etiology of the arrhythmia was not determined; however, imbalances in autonomic tone associated with trauma and/or direct trauma to the heart were hypothesized. Key clinical message: This report indicates a possible role of imbalances in autonomic tone due to trauma in the development of paroxysmal atrial fibrillation and suggests that it should be a differential diagnostic consideration in patients with atrial fibrillation following trauma. Primary treatment of atrial fibrillation may not be needed in these cases if the ventricular response rate is not rapid, or if there is spontaneous conversion to sinus rhythm.
Fibrillation atriale paroxysmique chez un chien présenté avec des blessures au cou. Une femelle braque allemand stérilisée âgée de 12 ans a subi des blessures extensives de morsure autour du cou. À la présentation, une fibrillation atriale fut identifiée avec un rythme de réponse ventriculaire rapide de 300 battements par minute (bpm). Le rythme de réponse ventriculaire diminua à 130 bpm à la suite de l'administration d'hydromorphone et d'oxygène. Sur la base du rythme aucune thérapie antiarythmique ne fut initiée. Le rythme cardiaque était retourné au rythme sinusal lors de la première réévaluation 2 jours plus tard, et le chien est demeuré en rythme sinusal à toutes les évaluations subséquentes. Avec la résolution de l'arythmie, une fibrillation atriale paroxysmique fut suspectée. L'étiologie sous-jacente de l'arythmie ne fut pas déterminée; toutefois, des débalancements du tonus autonome associés avec un trauma et/ou un trauma directement au coeur furent émis comme hypothèses.Message clinique clé:Ce rapport mentionne un rôle possible de débalancements du tonus autonome dus à un trauma lors du développement de fibrillation atriale paroxysmique et suggère que cela devrait être un diagnostic différentiel à considérer chez les patients avec fibrillation atriale à la suite d'un trauma. Le traitement initial de la fibrillation atriale pourrait ne pas être nécessaire dans ces cas si le rythme de la réponse ventriculaire n'est pas rapide ou s'il y a une conversion spontanée au rythme sinusal.(Traduit par Dr Serge Messier).
Subject(s)
Atrial Fibrillation , Dog Diseases , Animals , Anti-Arrhythmia Agents/therapeutic use , Atrial Fibrillation/etiology , Atrial Fibrillation/veterinary , Dog Diseases/diagnosis , Dog Diseases/drug therapy , Dogs , Female , Heart VentriclesABSTRACT
OBJECTIVES: The aim of this study was to assess outcomes in cats diagnosed with uroabdomen at a single referral centre. METHODS: Fifty-three cats diagnosed with uroabdomen at a veterinary teaching hospital were identified between June 2003 and September 2016. Data collected included signalment, presenting signs, aetiology, location of rupture, presence of concurrent injury, outcome of urine culture, presence of uroliths and packed cell volume (PCV)/creatinine/potassium levels at presentation. Cats managed medically and surgically were included, and the use of urinary catheters, cystotomy tubes and abdominal drains were recorded. It was determined if patients survived to discharge or if they were euthanased or died. RESULTS: Seventy-four percent (n = 39) of cats survived to discharge. Elevations in creatinine (P = 0.03) were shown to be significantly correlated with survival to discharge. Sex, age, location of rupture, presence of uroliths, outcome of urine culture, presence of concurrent injury, potassium at presentation and PCV at presentation were not associated with survival to discharge. There was no difference in survival between cats that were medically or surgically managed. CONCLUSIONS AND RELEVANCE: Cats that develop uroabdomen have a good chance of survival. Electrolyte and biochemistry values should be assessed at the time of presentation, in addition to the presence of concurrent injury.
Subject(s)
Cat Diseases , Patient Discharge , Animals , Cats , Hospitals, Animal , Hospitals, Teaching , Retrospective Studies , Rupture/veterinaryABSTRACT
BACKGROUND: Infectious canine hepatitis is a rarely encountered disease, that is caused by Canine Adenovirus-1. Clinical signs can vary dramatically, and neurological signs are rarely seen. Neurological manifestation of this disease is rarely reported in the veterinary literature. CASE PRESENTATION: A 5-week-old, male entire Husky cross puppy presented for a one-day history of abnormal neurological behaviour (circling, ataxia, vocalization and obtund mentation). The puppy was euthanized shortly after presentation due to rapid deterioration. Histopathology raised concerns for Canine Adenovirus 1 (CAdV-1) based on vasculitis in the brain and intranuclear inclusion bodies in endothelial cell and hepatocytes; immunohistochemistry on brain tissue confirmed CAdV-1 infection. CONCLUSIONS: This report discusses possible routes of infection and manifestations of adenovirus infections causing neurologic signs. It also provides a timely reminder that CAdV-1 should be considered a differential in unvaccinated dogs that present with neurological signs. Further studies are required to better understand the neurotrophic tendencies of this virus.
