1.
Med. intensiva (Madr., Ed. impr.)
; 47(2): 127-127, feb. 2023. ilus
Article
in English
| IBECS
| ID: ibc-215041
2.
3.
Med. intensiva (Madr., Ed. impr.)
; 45(6): 375-380, Agosto - Septiembre 2021.
Article
in English
| IBECS
| ID: ibc-222363
4.
Med Intensiva (Engl Ed)
; 45(6): 379-380, 2021.
Article
in English
| MEDLINE
| ID: mdl-34294236
Subject(s)
COVID-19/complications , Hypoxia/etiology , Oxygen/administration & dosage , Prone Position , SARS-CoV-2 , Extracorporeal Membrane Oxygenation , Humans , Hypertension, Pulmonary/etiology , Hypoxia/blood , Hypoxia/physiopathology , Hypoxia/therapy , Male , Middle Aged , Oxygen/blood , Oxygen Inhalation Therapy , Partial Pressure , Respiration, Artificial , Supine Position , Unnecessary Procedures
5.
6.
Int J Endocrinol
; 2014: 847827, 2014.
Article
in English
| MEDLINE
| ID: mdl-24744784
ABSTRACT
Diabetic cardiomyopathy is defined as a ventricular dysfunction initiated by alterations in cardiac energy substrates in the absence of coronary artery disease and hypertension. Hyperglycemia, hyperlipidemia, and insulin resistance are major inducers of the chronic low-grade inflammatory state that characterizes the diabetic heart. Cardiac Toll-like receptors and inflammasome complexes may be key inducers for inflammation probably through NF-κB activation and ROS overproduction. However, metabolic dysregulated factors such as peroxisome proliferator-activated receptors and sirtuins may serve as therapeutic targets to control this response by mitigating both Toll-like receptors and inflammasome signaling.