[Association of pseudochylothorax and pleural tuberculosis. Apropos of a case]. / Association pseudochylothorax et tuberculose pleurale. A propos d'une observation.

The authors report a case of pseudochylothorax revealed by pleural tuberculosis. Classically, pseudochylothorax is a late complication of chronical and calcified pleurisy especially sequellae of tuberculosis treated by induced pneumothorax. Treatment consist on thoracentesis or aspiration with frequent remove. Surgical decortication can be useful. Specific chemotherapy is only necessary in the patients in whom tuberculosis is present or never treated with antibiotics.

[Alcoholic ketoacidosis. Apropos of a case with coma]. / Acidocétose alcoolique. A propos d'un cas avec coma.

Alcoholic ketoacidosis is rare and must be considered in the differential diagnosis of any alcoholic patient with acidosis. The pathogenesis is discussed on the basis of one case. It is easily reversible by simple saline plus glucose infusion.

[Campylobacter fetus ssp fetus septicemia associated with a colonic abscess]. / Septicémie à Campylobacter fetus ssp fetus associée à un abcés colique.

Campylobacter is considered to be an opportunistic agent. The authors relate an unusual case with Campylobacter fetus ssp fetus (CF) septicemia and colic abscess. Human Campylobacteriosis is presumed to be a food-born disease related to contaminated animal products such as milk or meat. In some cases CF may be transmitted by drinking water or by fecal soiling via the hands. Conventional treatment uses macrolides with decrease the duration of diarrhea and reduce the fecal excretion of CF. Macrolides are ineffective in CF septicemias. In such cases aminoglycosides seem to be the drug of choice.

[Cholestatic hepatitis induced by the amoxicillin-clavulanic acid combination. A case and review of the literature]. / Hépatite cholestatique due à l'association amoxicilline-acide clavulanique. Un cas et revue de la littérature.

We report the case of a patient who developed jaundice after receiving amoxicillin-clavulanic acid for 7 days. Laboratory features were consistent with acute cholestatic hepatitis. Histopathological examination of a liver specimen showed cholestasis. Complete recovery occurred within 2 months after withdrawal of the drug. Analysis of the 24 reported cases of amoxicillin-clavulanic acid induced hepatitis revealed a predominantly cholestatic syndrome occurring soon after drug administration. In all cases, hepatic dysfunction disappeared within 1 to 3 months after discontinuation of the drug. Because of the small number of cases in contrast with the widespread use of this drug, associated with blood hypereosinophilia or eosinophilic infiltration of portal triads in some cases, a hypersensitivity phenomenon is suggested.