Your browser doesn't support javascript.
loading
Show: 20 | 50 | 100
Results 1 - 2 de 2
Filter
Add more filters










Database
Language
Publication year range
1.
Article in Russian | MEDLINE | ID: mdl-10852056

ABSTRACT

The bacteriological analysis of 302 children has revealed that dysbacteriosis with the increased content of hemolytic Escherichia in the intestine develops more frequently in the presence of the deficiency of bifido- and lactobacteria. The development of other kinds of dysbacteriosis with the increased content of different opportunistic enterobacteria, staphylococci, enterococci and fungi, as well as dysbacteriosis with the decreased content of Escherichia, does not practically depend on the deficiency of bifido- and lactobacteria. In patients with the increased content of Escherichia an increase in the content of opportunistic enterobacteria, staphylococci and fungi is observed more frequently than in patients with the low content of Escherichia.


Subject(s)
Bacterial Infections/microbiology , Intestinal Diseases/microbiology , Urban Population , Adult , Bacteria/isolation & purification , Child , Colony Count, Microbial , Feces/microbiology , Humans , Moscow
2.
Article in Russian | MEDLINE | ID: mdl-9825493

ABSTRACT

The examination of 52 patients with different forms of intestinal dysbacteriosis revealed a considerable decrease in the indices of immunity to endotoxin of gram-negative bacteria. In particular, a decrease in the content of antibodies to chemotype Re glycolipid, as well as in the content of polymorphonuclear leukocytes binding antibodies in the blood stream of the examined patients and leukocytes capable of binding endotoxin in endotoxin-treated blood smears, was registered, which was indicative of the loss of endotoxin binding reserves by leukocytes.


Subject(s)
Antibodies, Bacterial/biosynthesis , Endotoxins/immunology , Gram-Negative Bacterial Infections/immunology , Immunity/physiology , Intestinal Diseases/immunology , Case-Control Studies , Humans
SELECTION OF CITATIONS
SEARCH DETAIL
...