ABSTRACT
We report a 29-year-old man with a unique presentation of vasculitis as acute unilateral subdural effusion and meningoencephalitis. Magnetic resonance imaging showed a brainstem lesion that spread to the thalamus over time. There were no systemic features of vasculitis other than a positive pathergy test. Histopathological examination from the pathergy site showed neutrophilic infiltrate and leucocytoclastic vasculitis. The condition was steroid responsive and he remained in remission at two years' follow-up. The anatomy of the brainstem lesion, absence of other inflammatory and infective conditions on evaluation suggests a vasculitic pathology either as primary central nervous system angiitis or as neurological presentation of systemic vasculitis like Behetaet's disease although the international diagnostic criteria for Behetaet's were not fulfilled.
Subject(s)
Subdural Effusion/etiology , Vasculitis/complications , Adult , Brain Stem/pathology , Humans , Magnetic Resonance Imaging/methods , Male , Subdural Effusion/pathology , Tomography, X-Ray Computed/methodsABSTRACT
A 52-year-old male with right carotid body tumor underwent direct percutaneous glue (n-butylcyanoacrylate [NBCA]) embolization. Several hours later, he developed left hemiparesis from embolization of the polymerized glue cast. Migration of glue during percutaneous tumor embolization is presumed to occur only in the liquid state, which may lead to stroke or cranial nerve deficits. To the best of our knowledge, this is the first report of delayed glue embolization from a treated hypervascular tumor of the head and neck.
Subject(s)
Carotid Body Tumor/therapy , Cyanoacrylates/adverse effects , Embolization, Therapeutic/methods , Foreign-Body Migration/complications , Stroke/etiology , Carotid Body Tumor/blood supply , Cyanoacrylates/administration & dosage , Enbucrilate , Humans , Injections, Intralesional , Male , Middle AgedABSTRACT
We report a case of variant origin of superior thyroid artery (STA), occipital artery (OA) and ascending pharyngeal artery (APA) from the cervical segment of internal carotid artery (ICA) in a 63-year-old male patient, who presented with acute subarachnoid hemorrhage. Four-vessel angiography showed asymptomatic occlusion of ICA giving origin to the anomalous branches. There was common origin of the STA and the OA. The APA originated from the OA. We did not find any other description in the literature on variant origin of STA from the cervical segment of ICA in the presence of a well-developed external carotid artery.
Subject(s)
Carotid Artery, Internal/abnormalities , Occipital Lobe/blood supply , Pharynx/blood supply , Thyroid Gland/blood supply , Carotid Artery, External/diagnostic imaging , Carotid Artery, Internal/diagnostic imaging , Cerebral Angiography/methods , Humans , Male , Middle Aged , Subarachnoid Hemorrhage/complicationsABSTRACT
OBJECT: Vein of Galen malformations (VGMs) are extremely rare intracranial lesions. Clinical presentation and management strategies vary significantly in different areas of the world. The authors report their experience in evaluation, management, and long-term follow up of these lesions in India. METHODS: Between October 1983 and June 2003, 25 patients with VGMs were referred to the authors' institution for evaluation and management. Ten children younger than 2 years of age presented with rapidly increasing head size as the chief complaint. Among 11 children 2 years of age or older, the most common presenting symptom was chronic headache. Four patients who presented during adulthood had chronic headache for many years before presentation. Angiographic evaluation of the lesion was performed in 21 patients. Fifteen patients were treated using endovascular techniques. Injection of the embolic material was performed after induction of systemic hypotension when the flow in the fistula was high. Complete occlusion of the arteriovenous shunt could be achieved in two patients with vein of Galen aneurysmal dilation (100% of patients with this type of malformation) and in five of the six patients with the mural type of malformation (83%). Among patients with the choroidal type of malformation, complete obliteration of the shunt could be achieved in three patients. In three patients with high-flow choroidal malformations, embolization carried out in a single sitting resulted in shunt reduction of nearly 90%. These patients received clinical follow up. CONCLUSIONS: The authors' experience in evaluation and management of VGMs reveals that in areas of the world where access to dedicated specialist care is limited, the clinical presentation of VGMs can differ appreciably from the classic descriptions in the literature. Endovascular management of these lesions results in excellent angiographic and clinical results.
Subject(s)
Cerebral Veins/abnormalities , Embolization, Therapeutic , Intracranial Arteriovenous Malformations/diagnosis , Intracranial Arteriovenous Malformations/therapy , Adult , Age Factors , Child , Child, Preschool , Follow-Up Studies , Humans , India , Infant , Intracranial Arteriovenous Malformations/complications , Retrospective Studies , Treatment OutcomeSubject(s)
Leg , Magnetic Resonance Imaging , Muscle Weakness/diagnosis , Muscle Weakness/physiopathology , Spine/pathology , Adult , Humans , MaleABSTRACT
An incidental persistent falcine sinus was detected in an otherwise normal brain on MRI in a 12-year-old girl who underwent imaging after clinical suspicion of acute disseminated encephalomyelitis. The falcine sinus was associated with a hypoplastic posterior third of the superior sagittal sinus and a dominant straight sinus. Generally, atresia or hypoplasia of the straight sinus is associated with a persistent falcine sinus in postnatal life; otherwise, the falcine sinus disappears before birth. We discuss the embryological basis for such an association in this case.
