ABSTRACT
A case of postoperative cholecystitis in a 64 years old man is reported. This is a nosological entity characterized by gallbladder distension without any patent obstacle in the cystic duct and constancy of necrosis involving all the parietal layers. Clinical signs and symptoms are aspecific. Mortality rate is high and diagnosis has to be done quickly because the gallbladder necrosis makes cholecystectomy compulsory on such patients.
Subject(s)
Cholecystitis/surgery , Postoperative Complications/surgery , Acute Disease , Cholecystectomy , Cholecystitis/etiology , Cholecystitis/pathology , Gallbladder/pathology , Humans , Male , Middle Aged , Necrosis/pathology , Necrosis/surgery , Postoperative Complications/etiology , Postoperative Complications/pathologyABSTRACT
Primary malignant hepatic tumors are relatively common and the possibility of radical surgical treatment in the initial stages has modified our clinical approach therefore stressing the validity of a correct screening and follow-up of this pathology. The Authors present their clinical experience in the field enriched by the use of ultrasonography and conclude that this represents a highly effective diagnostic technique whereas it is less reliable in the determination of the nature of the lesion.
Subject(s)
Adenoma, Bile Duct/diagnosis , Bile Duct Neoplasms/diagnosis , Carcinoma, Hepatocellular/diagnosis , Liver Neoplasms/diagnosis , Ultrasonography , Adenoma, Bile Duct/pathology , Bile Duct Neoplasms/pathology , Carcinoma, Hepatocellular/pathology , Diagnosis, Differential , Humans , Liver Neoplasms/pathology , Neoplasm Staging , Preoperative CareABSTRACT
Two uncommon cases of Brunner gland hyperplasia are reported. Both presented clinical symptoms simulating gastrointestinal disease caused by hyperacidity. In conjunction with clinical statistics, diagnosis based on aetiology was only formulated after a double-contrast radiological exam of the g-i tract. After treatment with antiacid and antisecretory drugs, remission of the clinical symptoms was obtained, and regression of the hamartomatous polypoid duodenal neoformation, thus confirming the hypothesis which states that hyperacidic gastric secretion is the main cause of Brunner gland hyperplasia.