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1.
ACS Appl Mater Interfaces ; 9(11): 10198-10202, 2017 Mar 22.
Article in English | MEDLINE | ID: mdl-28098971

ABSTRACT

Here we report another surprising feature of the methylammonium metal halide material family, the phototunability of the diode response of a heterojunction made of CH3NH3PbI3 and its close relative, CH3NH3SnI3. In the dark state the device behaves as a diode, with the Sn homologue acting as the "p" side. The junction is extremely sensitive to illumination. A complete reversal of the diode polarity, the first observation of its kind, is seen when the junction is exposed to red laser light of 25 mW/cm2 or larger power density. This finding opens up the possibility for a novel class of optoelectronic devices.

2.
Phys Rev Lett ; 97(6): 067206, 2006 Aug 11.
Article in English | MEDLINE | ID: mdl-17026201

ABSTRACT

The field dependence of the electron spin resonance in a helimagnet LiCu2O2 was investigated for the first time. In the paramagnetic state, a broad resonance line was observed corresponding to a g factor of 2.3. In the critical regime, around the paramagnetic to helimagnetic phase transition the resonance broadens and shifts to higher frequencies. A narrow signal is recovered at a low temperature, corresponding to a spin gap of 1.4 meV in zero field. A comprehensive model of the magnons is presented, using exchange parameters from neutron scattering [T. Masuda Phys. Rev. B 72, 014405 (2005)10.1103/PhysRevB.72.014405] and the spin anisotropy determined here. The role of the quantum fluctuations is discussed.

3.
Neurobiol Aging ; 27(2): 228-36, 2006 Feb.
Article in English | MEDLINE | ID: mdl-15894409

ABSTRACT

The pathological hallmarks of Alzheimer's disease (AD) consist of beta-amyloid plaques and neurofibrillary tangles in affected brain areas. The processes, which drive this host reaction are unknown. To determine whether an analogous host reaction to that occurring in AD could be induced by infectious agents, we exposed mammalian glial and neuronal cells in vitro to Borrelia burgdorferi spirochetes and to the inflammatory bacterial lipopolysaccharide (LPS). Morphological changes analogous to the amyloid deposits of AD brain were observed following 2-8 weeks of exposure to the spirochetes. Increased levels of beta-amyloid precursor protein (AbetaPP) and hyperphosphorylated tau were also detected by Western blots of extracts of cultured cells that had been treated with spirochetes or LPS. These observations indicate that, by exposure to bacteria or to their toxic products, host responses similar in nature to those observed in AD may be induced.


Subject(s)
Alzheimer Disease/microbiology , Amyloid beta-Peptides/metabolism , Borrelia/physiology , Neuroglia , Alzheimer Disease/etiology , Alzheimer Disease/metabolism , Animals , Bacterial Infections/metabolism , Bacterial Infections/pathology , Blotting, Western/methods , Brain/metabolism , Brain/microbiology , Brain/pathology , Cells, Cultured , Disease Models, Animal , Immunohistochemistry/methods , Lipopolysaccharides/toxicity , Neurofibrillary Tangles/metabolism , Neurofibrillary Tangles/pathology , Neuroglia/drug effects , Neuroglia/metabolism , Neuroglia/microbiology , Neurons/drug effects , Neurons/metabolism , Neurons/microbiology , Organ Culture Techniques , Rats , Rats, Sprague-Dawley , Spectrophotometry, Infrared/methods , Time Factors
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