[Newly occurring stenocardia: the clinical picture, diagnosis, treatment and prognosis]. / Vpervye voznikshaia stenokardiia: klinika, diagnostika, lechenie, prognoz.

The article analyses the results of treatment of 77 patients with primarily emerged stenocardia. Three variants of clinical course of illness were distinguished: primarily emerged stenocardia at the state of exertion, primarily emerged progressive stenocardia at the state of exertion, primarily emerged stenocardia at rest state. As for primarily emerged stenocardia at the state of exertion, the changes at the final part of QRS complex were disclosed in 56% of patients, and abnormalities in the mobility of the left ventricle walls in 32%. The contractive function of left ventricle was unchanged. The myocardial infarction developed in 8% of patients in the course of one month, and in 4% in the course of one year. Stenocardia attacks disappeared in 16% of patients. As for primarily emerged progressive stenocardia, the ECG changes were marked in 74% of cases. The whole sphygmic fraction in 65% of patients was 55% lower. The myocardial infarction developed in 12% of patients in the course of one month and in 10.7% in more remote period. 52% of patients with the primarily emerged stenocardia at rest had ECG changes. In ECG daily monitoring 33% of patients had characteristic changes of coronary artery spasm. Myocardial infarction occurred in 8% of patients in the course of the first year. Stenocardia attacks disappeared in 25% of patients.

[Invasive treatment methods in therapy and angiogenic sepsis]. / Invazivnye metody lecheniia v terapii i angiogennyi sepsis.

In 1850 patients with intravenous catheters, intravenous shunts and fistulas sepsis developed in 0.43%. Infection of the catheters and shunts was more frequent than sepsis development. The growth of microorganisms (Staphylococcus aureus, Gram-negative microbes) was noted during a bacteriological study of 300 removed catheters in 27.7%. Angiogenic sepsis was caused by Staphylococcus aureus. Peculiarities of the clinical picture of angiogenic sepsis were discussed. Its early diagnostic signs were described. Immunological changes in the patients with angiogenic sepsis were evaluated. The problems of pathogenesis and prevention of angiogenic sepsis were given due attention.