ABSTRACT
Tinnitus is a challenging condition to manage with no effective treatment. We report a case of lifelong tinnitus present in a patient who was cured with the surgical repair of his coarctation of aorta. The patient was a fit 37-year-old with coarctation of aorta, senserineural deafness and intractable tinnitus. He underwent a thoracotomy and tube by-pass of his coarctation of aorta. A complete relief of his tinnitus and improvement in his hearing was noted postoperatively and confirmed on audiograms. This is the first reported case of tinnitus due to coarctation of aorta relieved by surgical correction of the coarctation.
ABSTRACT
Click-evoked gross potentials were recorded from the round windows of 29 cats previously exposed to high-level sounds. The latency and amplitude of the gross neural components of these responses were determined and compared with the patterns of threshold shift measured in single auditory nerve fibers from the same 29 animals. Both of these electrophysiological measures were compared with the patterns of hair cell loss as seen in celloidin sections through the temporal bone. The correlations between single-unit abnormalities and cochlear pathology in these cases have been documented elsewhere. In this report, the correlations between gross-potential abnormalities and cochlear pathology are examined. The diagnostic potential of these correlations is discussed.
Subject(s)
Cochlea/physiology , Evoked Potentials, Auditory , Vestibulocochlear Nerve/physiology , Acoustic Stimulation , Animals , Cats , Hair Cells, Auditory/physiology , Hair Cells, Auditory, Inner/physiology , Neurons/physiology , Noise , Reaction TimeABSTRACT
The ear that is deaf and continues to discharge after radical or modified radical mastoidectomy remains an unsolved problem for the otologist. The temporal bones of 17 patients in whom open mastoidectomy had been done during their life time were serially sectioned and examined. Histological changes secondary to surgery and chronic infection are described. The study fails to reveal a specific cause for unhealed areas in the mastoid bowl. It is suggested that the unhealed areas are caused by the unhealthy physical environment for the surface epithelium.
