ABSTRACT
We present the case of a 59-year-old man, with exertional angina, atrial fibrillation, electrocardiographic and scintigraphic signs of inferior necrosis and anterior ischemia. An abnormally high take off of the right coronary artery, with anomalous intraaortic initial pathway, was objectified in the patient by means of angiography and magnetic resonance imaging. The patient did not have coronary atherosclerosis or intracoronary lesions.
Subject(s)
Coronary Vessel Anomalies/diagnosis , Myocardial Ischemia/diagnosis , Coronary Angiography , Coronary Vessel Anomalies/complications , Heart/diagnostic imaging , Humans , Magnetic Resonance Imaging , Male , Middle Aged , Myocardial Ischemia/etiology , Myocardium/pathology , Radiopharmaceuticals , Technetium Tc 99m Sestamibi , Tomography, Emission-Computed, Single-PhotonSubject(s)
Brucellosis , Pericarditis/etiology , Female , Humans , Male , Middle Aged , Pericardial Effusion/microbiology , Pericarditis/microbiologySubject(s)
Heart Failure/etiology , Osteitis Deformans/complications , Humans , Male , Middle Aged , Osteitis Deformans/diagnosisABSTRACT
In a previous study we showed that the lesions of non-bacterial thrombotic endocarditis induced by means of implantation of a catheter in the left ventricle (LV) of the rabbit, undergo inner connectivization and surface endothelialization, which are completed within 2-3 months. In the present study we have investigated whether these histological changes lead to a variation in susceptibility to infective endocarditis (IE). After studying two control groups, we compared the incidence of IE in four groups of 15 rabbits each, inoculated with Streptococcus mitis I, 10, 35 and 70 days after implantation of a catheter in the LV. The frequency of infection was shown to be progressively reduced from 100% to 26.7%. This demonstrates that endothelialization of the catheter and the sterile vegetations protect the animals from IE.
Subject(s)
Cardiac Catheterization/adverse effects , Endocarditis, Bacterial/etiology , Animals , Catheters, Indwelling/adverse effects , Disease Susceptibility , Endocarditis, Bacterial/pathology , Female , Heart Valves/pathology , Heart Ventricles , Male , Myocardium/pathology , Rabbits , Streptococcal Infections/etiology , Streptococcal Infections/pathology , Streptococcus/pathogenicity , Time FactorsSubject(s)
Meningitis, Meningococcal/immunology , Antibodies, Bacterial/immunology , Bacterial Vaccines/immunology , Carrier State/immunology , Complement System Proteins/deficiency , Disease Outbreaks/epidemiology , Humans , Meningitis, Meningococcal/epidemiology , Neisseria meningitidis/classification , Neisseria meningitidis/pathogenicity , SerotypingSubject(s)
Heroin Dependence/complications , Myelitis, Transverse/etiology , Myelitis/etiology , Acute Disease , Adolescent , Humans , MaleSubject(s)
Abscess/etiology , Heroin Dependence/complications , Spinal Cord Compression/etiology , Adult , Epidural Space , Humans , Male , Paraplegia/etiologyABSTRACT
Two patients who had a huge pericardial effusion of at least 9 and 14 years' duration caused by cardiac toxoplasmosis are reported. Toxoplasma gondii were seen in the pericardial fluid, and serologic evidence also demonstrated the activity of the infection. These cases illustrate both the need to exclude toxoplasmosis in chronic pericardial effusion of unknown cause and the possibility of seeing toxoplasma in the pericardial fluid of patients with active toxoplasmic pericarditis. Moreover, in endemic areas, cardiac toxoplasmosis may not be an exceptional cause of chronic pericardial effusion.
