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1.
Opt Lett ; 48(7): 1578-1581, 2023 Apr 01.
Article in English | MEDLINE | ID: mdl-37221714

ABSTRACT

We propose a scheme for the creation of stable optical Ferris wheel (OFW) solitons in a nonlocal Rydberg electromagnetically induced transparency (EIT) medium. Depending on a careful optimization of both the atomic density and the one-photon detuning, we obtain an appropriate nonlocal potential provided by the strong interatomic interaction in Rydberg states that can perfectly compensate for the diffraction of the probe OFW field. Numerical results show that the fidelity remains larger than 0.96, while the propagation distance has exceeded 160 diffraction lengths. Higher-order OFW solitons with arbitrary winding numbers are also discussed. Our study provides a straightforward route to generate spatial optical solitons in the nonlocal response region of cold Rydberg gases.

2.
Chem Commun (Camb) ; 58(91): 12680-12683, 2022 Nov 15.
Article in English | MEDLINE | ID: mdl-36286612

ABSTRACT

2-Styrylthiophene-based donor-acceptor linear conjugated polymers with tunable cyano substituents are atom-economically obtained via direct C-H arylation for platinum-free photocatalytic hydrogen production, affording a HER of up to 9.79 mmol h-1 g-1.

3.
Zhongguo Zhong Yao Za Zhi ; 33(7): 813-5, 2008 Apr.
Article in Chinese | MEDLINE | ID: mdl-18589789

ABSTRACT

OBJECTIVE: The present study investigated the inhibitory effects of Chinese herb component sinapine on activity of acetylcholinesterase (AChE) in cerebral homogenate and blood serum of rats. METHOD: AChE was prepared from cerebral homogenate and blood serum of rats, respectively. Acetylcholinesterase activity assay kit and Chromatometry were used to detect the AChE activity. RESULT: Sinapine significantly inhibited AChE activity in vitro, with more effective on cerebral homogenate (IC50 3.66 micromol x L(-1)) than blood serum (IC50 22.1 micromol x L(-1)). CONCLUSION: Sinapine could significantly inhibit the cerebral AChE activity and may be a promising drug used for prevention and cure of Alzheimer's disease as a cholinesterase inhibitor.


Subject(s)
Acetylcholinesterase/blood , Acetylcholinesterase/metabolism , Brain/enzymology , Choline/analogs & derivatives , Cholinesterase Inhibitors/pharmacology , Alzheimer Disease/drug therapy , Alzheimer Disease/prevention & control , Animals , Brain/cytology , Choline/pharmacology , Choline/therapeutic use , Cholinesterase Inhibitors/therapeutic use , Rats
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