ABSTRACT
Nanoparticles of Zn1-xCuxS with Cu concentrations of x=0.0, 0.1, 0.2, 0.3 and 0.4 were prepared by a co-precipitation reaction method from homogeneous solutions of zinc and copper salts. Both the ZnS and ZnS:Cu nanoparticles excited at about 370 nm exhibits a broad green emission band peaking around 491 nm, which confirms the characteristic feature of Zn2+ as well as Cu2+ ions as luminescent centers in the lattice. The TEM micrographs showed spherical morphology for ZnS nanocrystals and the average size of the particles was estimated to be around 8.5 nm. At liquid nitrogen temperature, ESR signal characteristic of Cu2+ ions was observed in samples of all concentrations. ESR spectra analysis also indicated that Cu2+ ions enter the host lattice by replacing Zn2+ ions with distorted tetrahedral site symmetry.
Subject(s)
Copper/chemistry , Electron Spin Resonance Spectroscopy/methods , Metal Nanoparticles/chemistry , Sulfides/chemistry , Zinc Compounds/chemistry , Electromagnetic Fields , Ions , Light , Luminescence , Microscopy, Electron, Transmission/methods , Models, Chemical , Nanoparticles/chemistry , Nanotechnology/methods , Photochemistry/methods , Salts/chemistry , ZincABSTRACT
Neurological manifestations of skeletal fluorosis have been attributed to compressive radiculomyelopathy. Experimental fluorosis has shown evidence of myopathic changes. Data on human muscle pathology is very scanty. This study included 22 patients with established osteofluorosis. 16 of them showed only EMG changes of neurogenic muscle disease. Histochemistry and histopathology of muscle biopsies showed features of muscle atrophy, evidenced by 'type I' atrophy and 'type I' grouping. No myopathic changes were observed. It may be concluded that the primary changes are related to the nerve, with muscle being affected secondarily. There was no evidence of any primary muscle pathology due to fluorosis.
Subject(s)
Bone Diseases/pathology , Fluoride Poisoning/pathology , Muscle, Skeletal/pathology , Adenosine Triphosphatases/metabolism , Adult , Aged , Bone Diseases/chemically induced , Endemic Diseases , Female , Fluoride Poisoning/epidemiology , Humans , Male , Middle AgedABSTRACT
A case of acute traumatic interhemispheric subdural haematoma treated conservatively is reported. The controversies in its management and the usefulness of CT in planning the treatment are discussed.