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1.
Otol Neurotol ; 31(3): 404-8, 2010 Apr.
Article in English | MEDLINE | ID: mdl-20357630

ABSTRACT

OBJECTIVE: Despite most patients with single-sided deafness (SSD) after operation for acoustic neuroma (AN) perceiving a significant hearing handicap, less than 25% are interested in bone-anchored hearing aid (BAHA) implantation. We evaluated the treatment compliance of BAHA in SSD and the effect of BAHA on the subjective handicap. METHODS: Part 1: It was determined, from our first study, how many of the SDD patients after operation for AN (n = 59) and interested in BAHA (n = 14) had been implanted. Part 2: Of 23 BAHA-implanted patients with SSD due to various causes, including the implanted patients from the previous study, 21 answered a questionnaire on the BAHA treatment compliance and the subjective handicap with and without BAHA. RESULTS: Part 1: Of the 14 patients from our first study who were interested in implantation for BAHA, 11 had been implanted (18.6% of all 59 patients). Part 2: Of the 21 patients with SSD, 95% still used BAHA, and of these, 81% used it more than 8 hours a day. Of the patients, 90% considered BAHA a moderate to significant aid; it reduced the subjective hearing handicap from 7.4 to 2.3 arbitrary units on a visual analog scale. CONCLUSION: Despite the subjective handicap perception among most patients with SSD after AN surgery, less than 20% chose treatment with BAHA. Conversely, patients with SSD choosing implantation had high treatment compliance and felt that BAHA reduced their handicap. Thus, when selected by the patients after testing, BAHA is effective in SSD and results in a high patient compliance.


Subject(s)
Hearing Aids , Hearing Loss, Unilateral/surgery , Patient Compliance , Suture Anchors , Adult , Aged , Female , Humans , Male , Middle Aged , Patient Satisfaction , Prosthesis Implantation , Sound Localization/physiology , Statistics, Nonparametric , Surveys and Questionnaires , Treatment Outcome
2.
Auris Nasus Larynx ; 36(6): 721-4, 2009 Dec.
Article in English | MEDLINE | ID: mdl-19482450

ABSTRACT

Necrotizing sialometaplasia is a benign, self-limiting, inflammatory process involving salivary glands, commonly associated with tissue ischemia. Clinically, necrotizing sialometaplasia is most often found in the hard palate as a deep ulcer with raised, indurated edges that can be indolent. This, as well as the histopathologic findings of necrotizing sialometaplasia, can be confused with those of a malignant neoplasm. We report a rare case of necrotizing sialometaplasia in the larynx, probably initiated by an underlying malignant process. We suggest an aggressive diagnostic approach, when necrotizing sialometaplasia involves the larynx and no recent exposure to radiation, surgery or trauma has been recorded. Necrotizing sialometaplasia of the larynx should be regarded as secondary to malignancy until proven otherwise.


Subject(s)
Carcinoma, Adenosquamous/diagnosis , Laryngeal Diseases/diagnosis , Laryngeal Neoplasms/diagnosis , Sialometaplasia, Necrotizing/diagnosis , Adult , Biopsy , Carcinoma, Adenosquamous/pathology , Comorbidity , Diagnosis, Differential , Epiglottis/pathology , Female , Humans , Hypopharynx/pathology , Laryngeal Diseases/pathology , Laryngeal Neoplasms/pathology , Laryngoscopy , Magnetic Resonance Imaging , Neoplasm Invasiveness , Sialometaplasia, Necrotizing/pathology , Vocal Cords/pathology
3.
Otol Neurotol ; 29(3): 334-8, 2008 Apr.
Article in English | MEDLINE | ID: mdl-18277309

ABSTRACT

OBJECTIVE: Primary cholesteatoma of the external auditory canal (EACC) is a rare disease, characterized by osteonecrosis with formation of sequesters and ingrowth of keratinizing squamous epithelium in the bony EAC. The aetiology and pathogenesis are unknown, but an earlier study has demonstrated abnormal epithelial migration in such ears. The present study explored whether this interesting result can be reproduced. STUDY DESIGN: The epithelial migration in 10 ears with EACC was studied using the ink-dot method. Two ears with minor lesions were studied before treatment, and 8 ears were studied after operation or conservative treatment. The results were compared with the migration in 15 normal ears examined in a previous study using the same method. SETTING: Tertiary referral center. MAIN OUTCOME MEASURES: 1) Presence or absence of epithelial migration; 2) change of the normal centrifugal pattern; and 3) estimated migration speed compared with normal ears. RESULTS: A qualitatively normal centrifugal migration was present on intact (unaffected or healed) skin in all 10 ears with EACC and was only missing directly on crust-covered lesions. The estimated migration speed was similar to the speed in normal ears, that is, approximately 150 mum/d. CONCLUSION: Ears with EACC seem to have qualitatively and quantitatively normal epithelial migration except directly on crust-covered lesions. It is unlikely that an abnormal epithelial migration is involved in the pathogenesis of this disease. These observations have implications for EAC disorders with similar clinical features that involve bony invasion, including osteoradionecrosis.


Subject(s)
Cell Movement , Cholesteatoma/etiology , Cholesteatoma/pathology , Ear Canal/pathology , Epithelial Cells/pathology , Adult , Aged , Female , Humans , Male , Middle Aged , Osteonecrosis/etiology , Osteonecrosis/pathology , Wound Healing
4.
Scand J Infect Dis ; 38(4): 299-301, 2006.
Article in English | MEDLINE | ID: mdl-16709530

ABSTRACT

We report a case of Lemierre's syndrome caused by Fusobacterium necrophorum and discuss characteristics of this potentially fatal condition which, though rare, may have a rising frequency. Familiarity with the signs and symptoms of Lemierre's syndrome is therefore of great importance.


Subject(s)
Fusobacterium Infections/physiopathology , Fusobacterium necrophorum/pathogenicity , Adult , Anti-Bacterial Agents/therapeutic use , Fusobacterium Infections/drug therapy , Fusobacterium necrophorum/drug effects , Fusobacterium necrophorum/isolation & purification , Humans , Male , Microbial Sensitivity Tests , Syndrome
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