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1.
Environ Health Perspect ; 112(15): 1557-63, 2004 Nov.
Article in English | MEDLINE | ID: mdl-15531442

ABSTRACT

Climate change may increase the frequency and intensity of ozone episodes in future summers in the United States. However, only recently have models become available that can assess the impact of climate change on O3 concentrations and health effects at regional and local scales that are relevant to adaptive planning. We developed and applied an integrated modeling framework to assess potential O3-related health impacts in future decades under a changing climate. The National Aeronautics and Space Administration-Goddard Institute for Space Studies global climate model at 4 degrees x 5 degrees resolution was linked to the Penn State/National Center for Atmospheric Research Mesoscale Model 5 and the Community Multiscale Air Quality atmospheric chemistry model at 36 km horizontal grid resolution to simulate hourly regional meteorology and O3 in five summers of the 2050s decade across the 31-county New York metropolitan region. We assessed changes in O3-related impacts on summer mortality resulting from climate change alone and with climate change superimposed on changes in O3 precursor emissions and population growth. Considering climate change alone, there was a median 4.5% increase in O3-related acute mortality across the 31 counties. Incorporating O3 precursor emission increases along with climate change yielded similar results. When population growth was factored into the projections, absolute impacts increased substantially. Counties with the highest percent increases in projected O3 mortality spread beyond the urban core into less densely populated suburban counties. This modeling framework provides a potentially useful new tool for assessing the health risks of climate change.


Subject(s)
Geographic Information Systems , Greenhouse Effect , Models, Theoretical , Oxidants, Photochemical/poisoning , Ozone/poisoning , Population Dynamics , Public Health , Forecasting , Humans , Mortality/trends , New York/epidemiology , Seasons , Urban Population
2.
Am J Public Health ; 93(2): 209-14, 2003 Feb.
Article in English | MEDLINE | ID: mdl-12554571

ABSTRACT

The authors invoke a population health perspective to assess the distribution of environmental hazards according to race/ethnicity, social class, age, gender, and sexuality and the implications of these hazards for health. The unequal burden of environmental hazards borne by African American, Native American, Latino, and Asian American/Pacific Islander communities and their relationship to well-documented racial/ethnic disparities in health have not been critically examined across all population groups, regions of the United States, and ages. The determinants of existing environmental inequities also require critical research attention. To ensure inclusiveness and fill important gaps, scientific evidence is needed on the health effects of the built environment as well as the natural environment, cities and suburbs as well as rural areas, and indoor as well as outdoor pollutants.


Subject(s)
Environmental Exposure/statistics & numerical data , Ethnicity/classification , Health Status , Prejudice , Cost of Illness , Ethnicity/statistics & numerical data , Evaluation Studies as Topic , Female , Housing , Humans , Male , Risk Factors , Rural Health , Socioeconomic Factors , United States/epidemiology , Urban Health
3.
Environ Sci Technol ; 36(22): 4831-45, 2002 Nov 15.
Article in English | MEDLINE | ID: mdl-12487307

ABSTRACT

Atmospheric deposition is a significant loading pathway for polychlorinated dibenzo-p-dioxins and dibenzofurans (dioxin) to the Great Lakes. An innovative approach using NOAA's HYSPLIT atmospheric fate and transport model was developed to estimate the 1996 dioxin contribution to each lake from each of 5,700 point sources and 42,600 area sources in a U.S./Canadian air emissions inventory. These unusually detailed source-receptor modeling results show that deposition to each lake arises from a broad geographical region, with significant contributions from up to 2,000 km away. The source categories contributing most significantly to 1996 dioxin deposition appear to be municipal waste incineration, iron sintering, medical waste incineration, and cement kilns burning hazardous waste. Model-predicted air concentrations and deposition fluxes were consistent with ambient measurement data, within the uncertainties in each, but there may be a moderate tendency toward underestimation using midrange emissions estimates. The most likely reason for this tendency appears to be missing or underestimated emissions sources, but in-situ atmospheric formation of octachlorinated dibenzo-p-dioxin (OCDD) and heptachlorinated dibenzo-p-dioxin (HpCDD) may have also contributed. Despite uncertainties, the findings regarding the relative importance of different sources types and source regions appear to be relatively robust and may be useful in prioritizing pollution prevention efforts.


Subject(s)
Air Pollutants/analysis , Benzofurans/analysis , Models, Theoretical , Polychlorinated Dibenzodioxins/analogs & derivatives , Polychlorinated Dibenzodioxins/analysis , Soil Pollutants/analysis , Air Movements , Databases, Factual , Dibenzofurans, Polychlorinated , Great Lakes Region , Hazardous Waste
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