ABSTRACT
INTRODUCTION: Our aim was to analyze the correlation between growth status in height and chronological age, carpal maturation, cervical maturation, and dental maturation, and assess the diagnostic performance of Demirjian's dental maturation as an indicator of the pubertal growth spurt, through a retrospective longitudinal study. METHODS: Records of 60 Canadian patients obtained from the Burlington Growth Centre, which included height and weight charts and a set of x-rays at 6 points in time, were analyzed. The images at each point in time included 1 hand and wrist radiograph, a lateral cephalometric x-ray, and one 45° oblique cephalometric radiograph of each side, which were analyzed using the methods of Fishman, Baccetti, and Demirjian on the mandibular left and right second molars, respectively. The onset of the pubertal growth peak in height (distance to growth peak [DGP]) was identified, and the correlation between methods with DGP was assessed. RESULTS: High levels of correlation were obtained between the methods of Fishman, Baccetti, and Demirjian with DGP. The cutoff point between prepubertal and postpubertal stages was F stage for women and G stage for men, with statistically significant levels of sensitivity and specificity for the test. CONCLUSIONS: The use of the method of Demirjian applied to mandibular second molars is plausible as a predictor of the occurrence of the DGP for the studied population.
Subject(s)
Age Determination by Skeleton , Tooth Calcification , Bone Development , Canada , Female , Humans , Longitudinal Studies , Male , Molar , Retrospective StudiesABSTRACT
La displasia dentinaria (DD) es una condición autosómica dominante que afecta tanto los dientes temporales como los permanentes. En ella se observan piezas con defectos en la forma de sus raíces, parcial o completa obliteración de las cámaras pulpares, y predisposición para formar abscesos y quistes sin una causa previa. Se describe en la población en una proporción de 1 cada 100.000 pacientes. Esta anomalía fue descrita por primera vez en 1920, pero en 1939 se lo denominó como DD y luego, en 1957, Zellner se percató de su predisposición hereditaria por lo que consideró esta lesión como un síndrome de malformación mesodérmica. Histológicamente, el esmalte y la dentina de la corona aparecen normales, las cámaras pulpares se observan de forma semilunar y casi completamente obliteradas radiográficamente. En un artículo realizado se pudo concluir que su posible etiología sería un defecto en el componente epitelial, donde la invaginación de la vaina epitelial de Hertwig ocurre tempranamente, resultando en una raíz atrofiada con cámara y conductos radiculares obliterados.
