ABSTRACT
BACKGROUND: Infantile haemangiomas are the most common soft tissue tumours of infancy and childhood, with an incidence rate of 4-10 %. These lesions commonly present between 2 and 10 weeks after birth and characteristically involute around 8-10 years of age. Most lesions occurring in the scalp are managed conservatively, with surgery reserved for those with complications. CASE DESCRIPTION: A 5-year-old girl presented with a swelling over the left occiput noticed since birth, with a gradual increase in size and rapid progression since the last 1 year. She had undergone six sessions of intralesional steroids over the past 3 years. However, recent MRI and CT scans showed an increase in the swelling size with full-thickness erosion of the underlying calvarium without neuroparenchymal involvement. The patient underwent surgical excision of the lesion with an excellent outcome. Our literature review revealed only a single case of haemangioma which arose from the scalp and caused significant underlying calvarial erosion and intracranial extension. CONCLUSION: We present a case of an atypical infantile haemangioma arising in the subgaleal plane causing full-thickness erosion of the underlying calvarium.
Subject(s)
Head and Neck Neoplasms/pathology , Hemangioma/pathology , Scalp/pathology , Skin Neoplasms/pathology , Skull/pathology , Child, Preschool , Female , Head and Neck Neoplasms/complications , Head and Neck Neoplasms/congenital , Hemangioma/complications , Hemangioma/congenital , Humans , Skin Neoplasms/complications , Skin Neoplasms/congenitalABSTRACT
Spontaneous resolution of traumatic chronic subdural haematoma (CSDH) has been reported in literature. However, those with non traumatic CSDH are exceedingly rare and none reported with continued antiplatelet therapy where it itself is an aetiological agent for development of non traumatic CSDH. A 50-year-old male presented to us with a non haemorrhagic cerebellar infarct with a concomitant CSDH without history of any trauma. Patient's PT/INR, Bleeding time and Clotting time were normal. Patient was started on antiplatelet therapy (Tablet Aspirin 150 mg OD) for the acute infarct. MR Brain at 1 month showed an increased size of CSDH. However patient denied surgical evacuation hence we continued conservative line of management, however we continued anti-platelet therapy with close neurological and coagulation profile monitoring that remained within normal range throughout the period of observation. CT at 5(th) month showed complete resolution of CSDH. Patient was on antiplatelet drugs throughout the period of observation. Our case argues about the role of antiplatelet therapy in patients with CSDH with contrary lesions requiring anticoagulation.
