Subject(s)
Facial Neuralgia/classification , Facial Pain/classification , Terminology as Topic , Diagnosis, Differential , Facial Neuralgia/diagnosis , Facial Neuralgia/etiology , Facial Pain/diagnosis , Facial Pain/etiology , Humans , Temporomandibular Joint Disorders/complications , Temporomandibular Joint Disorders/diagnosisABSTRACT
The results of 20 years of investigation in the light of current literature data are indicative of the trigeminal neuralgia being a separate disease with its specific etiology and pathogenesis. In 93 to 97% of the patients compression of a trigeminal branch or radicle were primary causes of the disease. This provided a basis for the secondary allogenic system of paroxysmal type which explains the high efficacy of a number of antiepileptic drugs. With a lack of a steady effect of a conservative treatment, a surgical decompression of the trigeminal branch or radicle is a method of choice.
Subject(s)
Facial Neuralgia/diagnosis , Trigeminal Neuralgia/diagnosis , Diagnosis, Differential , Facial Neuralgia/etiology , Humans , Nerve Compression Syndromes/complications , Nerve Compression Syndromes/diagnosis , Pain Measurement , Trigeminal Nerve Injuries , Trigeminal Neuralgia/etiologyABSTRACT
On the basis of analyzing 800 observations on patients with neuralgias and prosopalgias, the authors identify special or paroxysmal neuralgias of the tunnel origin that are marked by a pathognomic clinical picture and require specific therapy. Another group of facial pains--prosopalgias proper--includes pains varying in the origin, clinical course and therapeutic approach. Diagnostic terms for describing neuralgias of the trigeminal nerve are reviewed, and the concept of their primarily central or primarily peripheral origin is proved to be untenable.
Subject(s)
Neuralgia/diagnosis , Cerebrovascular Disorders/complications , Face/innervation , Glossopharyngeal Nerve , Humans , Neuralgia/therapy , Pain , Trigeminal Nerve/blood supply , Trigeminal Neuralgia/diagnosis , Trigeminal Neuralgia/etiology , Vagus NerveABSTRACT
A comparative analysis of the effect from using the antiepileptic drugs tegretol, morpholep, sucsilep, trimethin, chlonazepam, and sodium valproate in 258 patients with trigeminal neuralgia was carried out. In half of the cases long-time follow-up observations were made. The results of the treatment were analyzed with reference to the time in which the therapeutic effect appeared and reached the maximum, as well as to the stability of the therapeutic remission obtained. Causes of failure of the therapy in some patients and the drugs' side-effects were analyzed.
Subject(s)
Anticonvulsants/therapeutic use , Trigeminal Neuralgia/drug therapy , Anticonvulsants/adverse effects , Carbamazepine/therapeutic use , Clonazepam/therapeutic use , Ethosuximide/therapeutic use , Follow-Up Studies , Humans , Morpholines/therapeutic use , Pyrrolidinones/therapeutic use , Valproic Acid/therapeutic useABSTRACT
A unique observation of an epileptic female examined in detail in a neurological clinic is presented. The patient was later hospitalized 3 times for disturbances of consciousness and extrapyramid disorders characterized by acute onset. An encephalographic examination carried out during such a state showed an epileptic status of atypic absence. The development of the Parkinsonian syndrome is regarded as a proof of the role of the caudate nucleus in the genesis of the petit mal epileptic status.
Subject(s)
Parkinson Disease, Secondary/complications , Status Epilepticus/diagnosis , Adult , Brain Diseases/complications , Brain Stem , Caudate Nucleus/physiopathology , Electroencephalography , Humans , Male , Status Epilepticus/complications , Status Epilepticus/physiopathologyABSTRACT
The paper is concerned with the results of a clinical electrophysiological, biochemical, histochemical, X-ray and morphological study of 289 patients with different vegetovascular and trophical disturbances, mainly of a regional character, in the facial area. Some clinical aspects and questions of the pathogenesis of these disturbances are discussed which are not sufficiently reflected in the literature. It is suggested that the form of the pterygopalatine ganglion is related to the incidence of the pathological process and the traits of vegetative and pain phenomena, accompanying the Sluder syndrome. Vegetovascular changes were revealed in the skin of the face and enzymatic changes in mucous membrane of the nose on the homolateral to the paresis side of the face in lesions of the facial nerve. Specific vegetative trophical changes were also found in a progressive hemiatrophy of the face, Barrakera-Simons disease, in the syndrome of facial hemistrophy due to lesions of the different vegetative and somatic structures of the nervous system.
Subject(s)
Face/innervation , Nervous System Diseases/diagnosis , Body Temperature , Facial Hemiatrophy/diagnosis , Facial Nerve , Facial Neuralgia/diagnosis , Lipodystrophy/diagnosis , Pain , Peripheral Nervous System Diseases/diagnosis , Pigmentation Disorders/diagnosis , Sweating , Syndrome , Trigeminal Neuralgia/diagnosis , Triglycerides/bloodABSTRACT
On the basis of clinical observations of 314 patients with neuralgia of the trigeminal nerve and a number of studies conducted on them (EEG, REG, tomography of the intraorbital canal, a morphological study on neurovascular fascicli removed during the operation of neurotomy and etc.) the authors discuss some problems of the etiology, pathogenesis, clinical picture and treatment of neuralgia of the trigeminal nerve.
