Subject(s)
Abdomen , Epilepsy/physiopathology , Pain/etiology , Adolescent , Child , Child, Preschool , Diagnosis, Differential , Epilepsy/diagnosis , Humans , Pain/diagnosis , SyndromeABSTRACT
Examination with the use of transient analysis is an objective method helping to assess the quality of vegetative regulation in children and its age-specific characteristics. The data obtained from studying the transient processes in clinically healthy children of different age groups may be used as the reference criteria in clinical studies of vegetative disorders, for establishing scientifically-based age-differentiated regimens for children with regard to adaptive potentialities of the body, as well as for the development of age periodization.
Subject(s)
Adaptation, Physiological , Autonomic Nervous System/physiology , Cardiovascular Physiological Phenomena , Adolescent , Age Factors , Cardiovascular System/innervation , Cerebrovascular Circulation , Child , Child, Preschool , Hemodynamics , Humans , Leg/blood supply , Myocardial Contraction , PostureSubject(s)
Hypothalamic Diseases/diagnosis , Adolescent , Child , Female , Humans , Male , Puberty , SyndromeABSTRACT
Owing to a high plasticity of the child's brain complete or partial compensation of a cerebral defect is possible. However an infection or a craniocerebral injury may interfere with the compensation of the premorbid defect of the nervous system: this was noted in 65% of children observed by the authors. Therefore, while specifying the syndromological structure of a disease one must take into account the time course of the premorbid defect. The compensation of that defect is hampered most often in children of the first year of the life. In these cases a loss of a part of acquired habits and appearance of earlier-age reflexes is noted. At the age of 2 to 5 years an aggravation of the atactic and hyperkinetic syndromes is observed, while at the age of over 5 years the negative time course of the premorbid defects is noted twice as rarer. Variants of the time course of the premorbid nervous disorders are specified. At the basis of reversible variants are disturbances on the part of hemodynamics and cerebrospinal fluid. This is confirmed by echo-EEG and REG findings. Persisting aggravation of the premorbid defect and appearance of new neurological symptoms are observed on development of hypoxic and metabolic disturbances. The nervous structures affected by perinatal hypoxia remain thenceforth to be especially sensitive to the effects of the latter. An analysis of the pathogenetic mechanisms of the premorbid defect decompensation may facilitate the realization of therapeutic and prophylactic measures.
Subject(s)
Brain Diseases/complications , Brain Injuries/complications , Communicable Diseases/complications , Ataxia/complications , Brain Concussion/complications , Child , Child, Preschool , Female , Fetal Hypoxia/complications , Humans , Hydrocephalus/congenital , Hyperkinesis/complications , Infant , Meningitis, Viral/complications , Mumps/complications , Pregnancy , Respiratory Tract Infections/complications , SyndromeABSTRACT
A clinico-electrophysiological characteristic of 56 children with the hemiatrophy syndrome is presented. The main clinical variants of hemiatrophy in children were distinguished. Some materials on the etiology and pathogenesis of this disorder are considered. The authors used electrophysiological methods in the study of this disorder: EEG, Echo EG, REG. The nozological entity of hemiatrophy the secondary symptomatical variants and features of the clinical development are discussed.
