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Vopr Pitan ; 77(3): 21-8, 2008.
Article in Russian | MEDLINE | ID: mdl-18669327

ABSTRACT

The analysis of the literature on communication of a hemostasis and vitamin C has allowed to assert, that its deficiency accelerates lipid peroxidation and reduces potential at porpoises, and it conducts to activation platelets, to acceleration of continuous intravascular blood coagulation of and to reduction in tolerance to thrombin. High dozes of vitamin C influences a hemostasis strengthens lipid peroxidation similarly to prooxidizers. Entering high dozes of vitamin C at the statuses menacing weith thromboses, it is desirable to supervise a level of markers of intravascular blood coagulation in plasma.


Subject(s)
Ascorbic Acid/metabolism , Blood Coagulation/physiology , Blood Platelets/metabolism , Homeostasis , Lipid Peroxidation/physiology , Platelet Activation/physiology , Animals , Ascorbic Acid/administration & dosage , Ascorbic Acid/adverse effects , Ascorbic Acid Deficiency/metabolism , Blood Coagulation/drug effects , Disseminated Intravascular Coagulation/chemically induced , Disseminated Intravascular Coagulation/metabolism , Humans , Lipid Peroxidation/drug effects , Platelet Activation/drug effects , Thrombin/metabolism , Thrombosis/chemically induced , Thrombosis/metabolism
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