Research on imaging method of driver's attention area based on deep neural network.

In the driving process, the driver's visual attention area is of great significance to the research of intelligent driving decision-making behavior and the dynamic research of driving behavior. Traditional driver intention recognition has problems such as large contact interference with wearing equipment, the high false detection rate for drivers wearing glasses and strong light, and unclear extraction of the field of view. We use the driver's field of vision image taken by the dash cam and the corresponding vehicle driving state data (steering wheel angle and vehicle speed). Combined with the interpretability method of the deep neural network, a method of imaging the driver's attention area is proposed. The basic idea of this method is to perform attention imaging analysis on the neural network virtual driver based on the vehicle driving state data, and then infer the visual attention area of the human driver. The results show that this method can realize the reverse reasoning of the driver's intention behavior during driving, image the driver's visual attention area, and provide a theoretical basis for the dynamic analysis of the driver's driving behavior and the further development of traffic safety analysis.

Protective effects of HTD4010, a Reg3a/PAP-derived peptide, in a mouse model of hypertriglyceridemic acute pancreatitis: Involvement of TLR4/NF-kappa B.

Acute pancreatitis (AP) is an inflammatory disorder of pancreas and common digestive diseases, effective drug for AP is few. As an analog of active center of Reg3α, the biological activities of HTD4010 are similar to Reg3α. This study aimed to explore whether HTD4010 could decrease the inflammatory response and pancreatic injury in hypertriglyceridemic-AP (HTG-AP), and explore underlying mechanisms. This study was shown that the administration of HTD4010 could decrease the inflammatory response, reduce acinar cell injury (both apoptosis and necroptosis) and damage of lung tissue in HTG-AP. Moreover, HTD4010 down-regulated the expression of TLR4 and NF-κB protein. These results showed that HTD4010 could alleviate the severity of AP possibly by TLR4/NF-κB signaling pathway in HTG-AP.