Estudio de la vascularización arterial del músculoflexor digitorum superficialis / Anatomical study of the arterial vascularization of the flexor digitorum superficialis muscle

El objetivo de nuestro estudio es describir la distribución de la vascularización del músculo flexor digitorum superficialis para optimizar sus indicaciones en Cirugía Reconstructiva de miembro superior. Disecamos 15antebrazos fijados según el método Thiel y coloreados mediante la inyección de látex en los vasos femorales. Centramos nuestro estudio en la disección del músculo flexor digitorum superficialis, seleccionando únicamente los pedículos vasculares que superan los 2 mm, valorando los resultados en función de sus relaciones anatómicas y de su longitud. El número total hallado de arterias nutrientes del vientre muscular fue de 219, localizándose en mayor porcentaje en el tercio medio del antebrazo. De los resultados obtenidos de nuestro estudio podemos deducir que las arterias cubital y cubital recurrente aportan la vascularización dominante. Medialmente, el músculo recibe ramas de la arteria cubital y cubital recurrente, en la parte profunda del vientre muscular. Lateral y proximalmente, recibe ramas de la arteria mediana, mientras que lateral y distalmente recibe ramas de la arteria radial, que penetran en la superficie del músculo (AU)

We present an anatomical study that describes the distribution of the muscular perforators of the flexor digitorum superficialis muscle. In this study we dissected15 forearms fixed according to Thiel method and coloured latex injection in the femoral vessels. The study was centered on the flexor digitorum superficialis muscle. Only muscular perforator arteries with diameters over2mm were selected. The vascular origin and length were also studied. In all cases, measurements were taken from the bicondyle line. The total number of arteries obtained from the muscle belly was 219, with the greatest percentage located in the half of the forearm. The principal vascular origin of the perforator arteries was the cubital artery. From the results obtained in our work, we can deduce that the ulnar and the ulnar recurrent arteries are the most dominant supply of the muscle. Medially, it receives many large branches from the ulnar recurrent and the ulnar artery. Laterally and proximally, it receives small branches from the median artery, and distally, it receives several small branches from the radial artery that enter the superficial surface of the muscle (AU)

Vascularización arterial del músculo flexor digitorum superficialis. Estudio anatómico y aplicaciones clínicas / Arterial vascularisation of the flexor digitorum superficialis muscle. Anatomical study and clinical aplications

El objetivo de nuestro estudio es describir la distribución de la vascularización del músculo flexor digitorum superficialis para optimizar sus indicaciones en cirugía reconstructiva de miembro superior. MATERIAL Y MÉTODOS. Disecamos 15 antebrazos fijados según el método Thiel y coloreados mediante la inyección de latex en los vasos femorales. Centramos nuestro estudio en la disección del músculo flexor digitorum superficialis, seleccionando únicamente los pedículos vasculares que superan los 2 mm, valorando los resultados en función de sus relaciones anatómicas y longitud. RESULTADOS. El número total de arterias nutrientes del vientre muscular era de 219, localizándose en mayor porcentaje en el tercio medio del antebrazo. CONCLUSIÓN. De los resultados obtenidos de nuestro estudio podemos deducir que la arteria cubital y cubital recurrente aportan la vascularización dominante. Medialmente recibe ramas de la arteria cubital y cubital recurrente, en la parte profunda del vientre muscular. Lateral y proximalmente recibe ramas de la arteria mediana, mientras que lateral y distalmente recibe ramas de la arteria radial, que penetran en la superficie del músculo (AU)

We present an anatomical study that describes the distribution of the muscular perforators of the flexor digitorum superficialis muscle. MATERIAL AND METHODS. In this study we dissected 15 forearms fixed according to Thiel method and coloured latex injection in the femoral vessels. The study was centered on the flexor digitorum superficialis muscle. Only muscular perforator arteries with diameters over 2mm were selected. The vascular origin and length were also studied. In all cases, measurements were taken from the bicondyle line. RESULTS. The total number of arteries obtained from the muscle belly was 219, with the greatest percentage located in the half of the forearm. The principal vascular origin of the perforator arteries was the cubital artery. ONCLUSION. From the results obtained in our work, we can deduce that the ulnar and the ulnar recurrent arteries are the most dominant supply of the muscle. Medially , it receives many large branches from the ulnar recurrent and the ulnar artery. Laterally and proximally, it receives small branches from the median artery, and distally, it receives several small branches from the radial artery that enter the superficial surface of the muscle (AU)

Enzymatic and morphological evaluation of the hepatic cytoprotective effect of somatostatin and octreotide during extended experimental liver preservation.

Cytoprotective effects on the liver of somatostatin (ST) and octreotide (OT) have been previously described in normothermic ischemia-reperfusion models. The purpose of this study was an enzymatic and morphological assessment of hepatic cytoprotective effects during extended cold storage. Rat livers were washed in situ via the portal vein with University of Wisconsin solution (UW) UW+ST, or UW+OT. After 24 or 48 hours of cold ischemia time (CIT), livers were reperfused for 2 hours via the portal vein with oxygenated KHB at 37 degrees C using a nonrecirculating ex situ isolated perfusion system. Levels of aspartate aminotransferase (AST), alanine aminotransferase (ALT), lactate dehydrogenase (LDH), and creatinine kinase (CK) were assessed in the perfusate during ex situ isolated reperfusion. After a 24-hour cold ischemia time (CIT) ALT, LDH, and CK levels were significantly lower (P < .05) in the UW+ST and the UW+OT livers than the UW livers. After 48-hour CIT, AST, ALT, LDH, and CK levels were significantly lower (P < .05) in the UW+ST and the UW+OT livers than the UW livers. Histopathological examination revealed mild differences after 24-hour CIT but an evidently less ischemically damage organ after 48-hour CIT. With the limitations of an in vitro model, ST and OT showed enzymatic and morphological effects during extended liver preservation.

Electrical impedance of the liver during experimental long-term liver preservation.

Measurements of electrical impedance were performed to assess ischemic damage in the rabbit liver during long-term preservation with University of Wisconsin (UW) or histidine-tryptophan-ketoglutarate (HTK) solution. The impedance was measured at a frequency of 200 Hz after in situ perfusion and after cold storage for 24 and 48 hours in UW or HTK solution (six livers per group). Z(200 Hz) was significantly higher (P < .01) after 48 compared with 24 hours of cold storage with both protection solutions without significant differences between the livers preserved with both solutions. Electrical impedance was observed to be a sensitive indicator of liver damage during long-term protection, showing similar preservation quality for both preservation solutions.