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Ann Urol (Paris) ; 25(4): 204-8, 1991.
Article in French | MEDLINE | ID: mdl-1746929

ABSTRACT

The addition of sucrose to drinking water of rats at the rate of 2.5 or 5 grams per 100 ml, for one month, induced hypercalciuria which appeared to be dependent on the degree of supplementation. In spite of these disorders, calcium deposits were not observed in treated animals. This protection against renal calculi was probably due to high urinary excretions of magnesium, phosphorus, zinc and copper. These lithogenesis inhibitors varied, like oxaluria and calciuria, in parallel with dietary sucrose intake.


Subject(s)
Dietary Carbohydrates/pharmacology , Kidney Calculi/etiology , Sucrose/pharmacology , Animals , Calcium/urine , Copper/urine , Dietary Carbohydrates/administration & dosage , Energy Intake , Kidney/metabolism , Magnesium/urine , Male , Organ Size , Oxalates/urine , Phosphorus/urine , Rats , Rats, Inbred Strains , Risk Factors , Sucrose/administration & dosage , Zinc/urine
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