A Review of Nitrogen-Doped Graphene Aerogel in Electromagnetic Wave Absorption.

Graphene aerogels (GAs) possess a remarkable capability to absorb electromagnetic waves (EMWs) due to their favorable dielectric characteristics and unique porous structure. Nevertheless, the introduction of nitrogen atoms into graphene aerogels can result in improved impedance matching. In recent years, nitrogen-doped graphene aerogels (NGAs) have emerged as promising materials, particularly when combined with magnetic metals, magnetic oxides, carbon nanotubes, and polymers, forming innovative composite systems with excellent multi-functional and broadband absorption properties. This paper provides a comprehensive summary of the synthesis methods and the EMW absorption mechanism of NGAs, along with an overview of the absorption properties of nitrogen-doped graphene-based aerogels. Furthermore, this study sheds light on the potential challenges that NGAs may encounter. By highlighting the substantial contribution of NGAs in the field of EMW absorption, this study aims to facilitate the innovative development of NGAs toward achieving broadband absorption, lightweight characteristics, and multifunctionality.

Carotid Geometry as a Predictor of In-Stent Neointimal Hyperplasia　- A Computational Fluid Dynamics Study.

BACKGROUND: Carotid angioplasty and stenting (CAS) is emerging as an alternative treatment for carotid stenosis, but neointimal hyperplasia (NIH) remains a drawback of this treatment strategy. This study aimed to evaluate the effect of variations of carotid bifurcation geometry on local hemodynamics and NIH.Methods and Results:Hemodynamic and geometric effects on NIH were compared between 2 groups, by performing computational fluid dynamics (CFD) simulations both on synthetic models and patient-specific models. In the idealized models, multiple regression analysis revealed a significant negative relationship between internal carotid artery (ICA) angle and the local hemodynamics. In the patient-derived models, which were reconstructed from digital subtraction angiography (DSA) of 25 patients with bilateral CAS, a low time-average wall shear stress (TAWSS) and a high oscillatory shear index (OSI) were often found at the location of NIH. Larger difference values of the OSI percentage area (10.56±20.798% vs. -5.87±18.259%, P=0.048) and ECA/CCA diameter ratio (5.64±12.751% vs. -3.59±8.697%, P=0.047) were detected in the NIH-asymmetric group than in the NIH-symmetric group. CONCLUSIONS: Changes in carotid bifurcation geometry can make apparent differences in hemodynamic distribution and lead to bilateral NIH asymmetry. It may therefore be reasonable to consider certain geometric variations as potential local risk factors for NIH.

Design of PVDF sensor array for determining airflow direction and velocity.

An airflow sensor comprised of an array of piezoelectric polyvinylidene fluoride (PVDF) cantilever sensors mounted on a sensor ring is fabricated. A fluid-solid-electric coupling model based on the finite element method is presented to obtain the mathematical relationship between the normal airflow velocity and the response voltage. According to the response voltages from all pieces of PVDF cantilevers in the array, the values of the airflow direction and the velocity can be calculated. Furthermore, to find a suitable algorithm for error calculations and to achieve high accuracy, a method of reducing the flow angle error ( Eαn,cal¯ ) and flow velocity error ( Δvn,cal¯ ) by extracting Um of the effective cantilevers can be established. The experimental results show that the maximum value of Eαn,cal¯ is 1.2° (at 270° with 11.1 m/s) and the minimum value of Eαn,cal¯ is 0.3° (at 135° with 11.1 m/s) based on the PVDF sensor array with eight cantilevers. Meanwhile, the maximum value of Evn,cal¯ is 3% (at 315° with 11.1 m/s), and the minimum value of Evn,cal¯ is 1.5% (at 360° with 11.1 m/s). In addition, under 20 random airflow angles at 8 m/s, the error range in airflow velocity is from 1.27% to 2.67%, the error range in airflow angle is from 0.34° to 1.24°, and the response time is 20 ms. Therefore, the proposed design for an airflow sensory ring array can be used to determine the airflow direction and velocity, and the airflow sensor can be miniaturized as a bionic antennae, which is mounted on the skin of a piezoelectric autonomous mobile robot for sensing and escaping from an attack of the natural enemy.

Mathematical modeling of atherosclerotic plaque destabilization: Role of neovascularization and intraplaque hemorrhage.

Observational studies have identified angiogenesis from the adventitial vasa vasorum and intraplaque hemorrhage (IPH) as critical factors in atherosclerotic plaque progression and destabilization. Here we propose a mathematical model incorporating intraplaque neovascularization and hemodynamic calculation with plaque destabilization for the quantitative evaluation of the role of neoangiogenesis and IPH in the vulnerable atherosclerotic plaque formation. An angiogenic microvasculature is generated by two-dimensional nine-point discretization of endothelial cell proliferation and migration from the vasa vasorum. Three key cells (endothelial cells, smooth muscle cells and macrophages) and three key chemicals (vascular endothelial growth factors, extracellular matrix and matrix metalloproteinase) are involved in the plaque progression model, and described by the reaction-diffusion partial differential equations. The hemodynamic calculation of the microcirculation on the generated microvessel network is carried out by coupling the intravascular, interstitial and transvascular flow. The plasma concentration in the interstitial domain is defined as the description of IPH area according to the diffusion and convection with the interstitial fluid flow, as well as the extravascular movement across the leaky vessel wall. The simulation results demonstrate a series of pathophysiological phenomena during the vulnerable progression of an atherosclerotic plaque, including the expanding necrotic core, the exacerbated inflammation, the high microvessel density (MVD) region at the shoulder areas, the transvascular flow through the capillary wall and the IPH. The important role of IPH in the plaque destabilization is evidenced by simulations with varied model parameters. It is found that the IPH can significantly speed up the plaque vulnerability by increasing necrotic core and thinning fibrous cap. In addition, the decreased MVD and vessel permeability may slow down the process of plaque destabilization by reducing the IPH dramatically. We envision that the present model and its future advances can serve as a valuable theoretical platform for studying the dynamic changes in the microenvironment during the plaque destabilization.