Recent Advance and Modification Strategies of Transition Metal Dichalcogenides (TMDs) in Aqueous Zinc Ion Batteries.

In recent years, aqueous zinc ion batteries (ZIBs) have attracted much attention due to their high safety, low cost, and environmental friendliness. Owing to the unique layered structure and more desirable layer spacing, transition metal dichalcogenide (TMD) materials are considered as the comparatively ideal cathode material of ZIBs which facilitate the intercalation/ deintercalation of hydrated Zn2+ between layers. However, some disadvantages limit their widespread application, such as low conductivity, low reversible capacity, and rapid capacity decline. In order to improve the electrochemical properties of TMDs, the corresponding modification methods for each TMDs material can be designed from the following modification strategies: defect engineering, intercalation engineering, hybrid engineering, phase engineering, and in-situ electrochemical oxidation. This paper summarizes the research progress of TMDs as cathode materials for ZIBs in recent years, discusses and compares the electrochemical properties of TMD materials, and classifies and introduces the modification methods of MoS2 and VS2. Meanwhile, the corresponding modification scheme is proposed to solve the problem of rapid capacity fading of WS2. Finally, the research prospect of other TMDs as cathodes for ZIBs is put forward.

Tubgcp3 is a mitotic regulator of planarian epidermal differentiation.

Tubgcp3/GCP3 (The centrosomal protein γ-tubulin complex protein 3) is a component of the γ-tubulin small complexes (γ-TuSCs) and γ-tubulin ring complexes (γ-TuRCs), which play critical roles in mitotic spindle formation during mitosis. However, its function in stem cell development has not been thoroughly elucidated. The planarian flatworm, which contains a large number of adult somatic stem cells (neoblasts), is a unique model to study stem cell lineage development in vivo. Here, we identified a homolog of Tubgcp3 in planarian Dugesia japonica, and found that Tubgcp3 is required for the maintenance of epidermal lineage. RNAi targeting Tubgcp3 resulted in tissue homeostasis and regeneration defect. Knockdown of Tubgcp3 reduced cell divisions and led to a loss of the mature epidermal cells. Our findings indicate that Tubgcp3 is a mitotic regulator and plays a crucial role in planarian epidermal differentiation.