[The effect of massive blood loss on the adrenaline-sensitive pool of the blood neutrophils]. / Vliianie massivnoi krovopoteri na adrenalinchuvstvitel'nyi pul neitrofilov krovi.

Experiments were conducted on models of immobilization stress and hemorrhagic shock in rabbits to study the changes in the ability of intravenously injected adrenaline (A) to induce blood neutrophilia due to the escape of parietal neutrophils into circulation. It is shown that immobilization stress, just like A infusion, leads to neutrophilia. In contrast, in hemorrhagic shock neutropenia develops and the response to A disappears. Such changes in massive blood loss are interpreted by the authors as transition of the marginal (physiological) adhesion of neutrophils in the vascular system to pathological adhesion characteristic of inflammatory reactions.

[Oxidative phosphorylation in pulmonary mitochondria after massive blood loss and restitution by autologous transfusion]. / Okislitel'noe fosforilirovanie v mitokhondriiakh legkikh pri massivnoi krovopotere i vospolnenii ee autokrov'iu.

A procedure is described for isolation and polarographic study of rabbit lung mitochondria exhibiting high respiratory control as well as coupling of oxidation and phosphorylation approaching the theoretical value. The procedure enabled to detect a reversible impairment of respiratory and phosphorylating functions of lung mitochondria under conditions of haemorrhagic shock, which was the most distinct within the first hours after blood reinfusion. Correlation was established between dynamics of alterations in mitochondrial energy production in lung tissue within posthaemorrhagic period and structure-functional characteristics of the lung surface system.