A modified repair technique for tricuspid incompetence in Ebstein's anomaly.

OBJECTIVE: A modified technique for tricuspid valve repair in Ebstein's anomaly restructures the valve mechanism at the level of the true tricuspid anulus by using the most mobile leaflet for valve closure without plication of the atrialized chamber. Midterm results of this therapeutic approach for patients with Ebstein's anomaly and tricuspid valve incompetence are reported. METHODS: Between October 1988 and April 1997, the incompetent tricuspid valve was repaired with our technique in 19 patients (12 female, 7 male; 2 to 54 years, mean 21 years). The indication for operation was congestive heart failure of various degrees in all patients. Tricuspid incompetence was grade II in two patients, grade III in 14, and grade IV in three. Associated congenital malformations were simultaneously repaired (interatrial communication in 18, ventricular septal defect in two, pulmonary stenosis in two, mitral valve prolapse in one). Follow-up ranged between 10 and 103 months (median 28 months) and was complete for all patients. RESULTS: There were no operative deaths. One patient with active endocarditis and pulmonary abscess died 2 months after the operation of recurrent sepsis; there were no late deaths. During follow-up, New York Heart Association functional class improved from 2.8 before the operation to 1.9 without recurrent cyanosis, and tricuspid incompetence decreased from a mean grade of 3.1 to one of 0.9, without any echocardiographic deterioration of the tricuspid valve function or right ventricular dilation. CONCLUSIONS: Our technique allows tricuspid valve repair in patients with Ebstein's anomaly, even in cases usually reserved for primary valve replacement, without late functional deterioration.

Hemodynamic changes during mechanical ventilation in infants and small children after open heart surgery.

The study was undertaken to clarify the hemodynamic effects of intermittent positive pressure ventilation (IPPV) and intermittent mandatory ventilation (IMV) with variation of the positive end-expiratory pressure (PEEP) from 5 to 15 mbar. The cardiac index (CI) was measured with thermodilation techniques in 30 infants who underwent open-heart surgery with extracorporeal circulation for various congenital heart lesions. The age of the patients varied from 6 to 28 months and body weight from 4 to 15 kg. During IPPV the changing of PEEP levels up to 5 mbar did not have any effect on Cl. Further increase in the PEEP to 10 and 15 mbar caused a significant decrease in Cl (from 2.6 to 2.0 l.min-1.m-2, p less than 0.05). The oxygen consumption (VO2) did not change significantly (135 ml.min-1.m-2 to 128 l.min-1.m-2, p greater than 0.5). A positive end-expiratory pressure exceeding 5 mbar caused a decrease of intrapulmonary veno-arterial blood shunting (QS/QT) from 12.3 to 7.1%; p less than 0.01), while PEEP at the level of 5 mbar did not affect this parameter. The alveolo-arterial oxygen gradient (AsDO2) also decreased from 182 to 135 torr (p less than 0.01) when PEEP was 10 and 15 mbar.(ABSTRACT TRUNCATED AT 250 WORDS)