[Organisms of the Campylobacter type in Barrett esophagus]. / Organismos tipo Campylobacter en el esófago de Barrett.

It is clear that CP is present in a higher or lower degree in different gastric-duodenum pathologies, especially in active superficial chronic gastritis, gastric ulcer and duodenum ulcer with gastric metaplasia. It is also found in atrophic chronic gastritis and, to a lesser extent, if it has intestinal metaplasia, as well as in some normal stomachs. It is not found in a histologically normal duodenum, nor in the oesophagus. As the fact that there was no publication on BE drew our attention, we set to make a retrospective research of CP of spinal metaplasia of distal oesophagus. Its presence proved to be high, 88% even in those cases with intestinal metaplasia and with ulcer of Barrett. We have used Gram coloration and Warthin Starry with Alcian-Blue and we have classified it within the degrees set by Marshall and Warren. We have also carried out a discussion on certain physiopathological facts, such as the presence of infiltrated PMN in all the cases, and its importance in keeping metaplasia, of ulcers of Barrett and its possible role in the development of adenocarcinoma.

Organismos tipo Campylobacter en el esófago de Barrett. / [Organisms of the Campylobacter type in Barrett esophagus]

It is clear that CP is present in a higher or lower degree in different gastric-duodenum pathologies, especially in active superficial chronic gastritis, gastric ulcer and duodenum ulcer with gastric metaplasia. It is also found in atrophic chronic gastritis and, to a lesser extent, if it has intestinal metaplasia, as well as in some normal stomachs. It is not found in a histologically normal duodenum, nor in the oesophagus. As the fact that there was no publication on BE drew our attention, we set to make a retrospective research of CP of spinal metaplasia of distal oesophagus. Its presence proved to be high, 88

even in those cases with intestinal metaplasia and with ulcer of Barrett. We have used Gram coloration and Warthin Starry with Alcian-Blue and we have classified it within the degrees set by Marshall and Warren. We have also carried out a discussion on certain physiopathological facts, such as the presence of infiltrated PMN in all the cases, and its importance in keeping metaplasia, of ulcers of Barrett and its possible role in the development of adenocarcinoma.

[Diagnosis and etiology of Barrett esophagus. Presence of gastrin secreting cells (G cells)]. / Diagnóstico y etiología del esofago de Barrett. Presencia de células secretoras de gastrina (G).

We present the patterns for the diagnosis, checking the clinical, radiological, endoscopical and histological data of 35 patients suffering from Barrett's Esophagus (BE) (columnar metaplasia lining the lower esophagus). The clinical characteristics are those of a severe esophagitis of long evolution, although metaplasia itself is asymptomatic, and its features depend on the inflammation degree. Radiology can bring out some data as GE reflux, hiatal hernia, ulcers or stricture, and perhaps double contrast may show any sign by means of which endobrachyesophagus (EBE) can be suspected. Endoscopy provides us with accurate data about EBE, ulcers, stricture and inflammation. Histology reveals the type of columnar metaplasia (junctional or cardial, gastric fundic, intestinal or specialized, or composite). Acquired or congenital etiology can be clarified by an immunohistochemical method, Peroxidase anti-Peroxidase (PAP), showing the presence of gastrin secretory cells (G cells) in the congenital cases.

Diagnóstico y etiología del esofago de Barrett. Presencia de células secretoras de gastrina (G). / [Diagnosis and etiology of Barrett esophagus. Presence of gastrin secreting cells (G cells)]

We present the patterns for the diagnosis, checking the clinical, radiological, endoscopical and histological data of 35 patients suffering from Barretts Esophagus (BE) (columnar metaplasia lining the lower esophagus). The clinical characteristics are those of a severe esophagitis of long evolution, although metaplasia itself is asymptomatic, and its features depend on the inflammation degree. Radiology can bring out some data as GE reflux, hiatal hernia, ulcers or stricture, and perhaps double contrast may show any sign by means of which endobrachyesophagus (EBE) can be suspected. Endoscopy provides us with accurate data about EBE, ulcers, stricture and inflammation. Histology reveals the type of columnar metaplasia (junctional or cardial, gastric fundic, intestinal or specialized, or composite). Acquired or congenital etiology can be clarified by an immunohistochemical method, Peroxidase anti-Peroxidase (PAP), showing the presence of gastrin secretory cells (G cells) in the congenital cases.

Measurement of intrahepatic pressure in alcoholic liver disease.

Intrahepatic pressure was measured in 172 subjects (148 patients with chronic alcoholic liver disease and 24 controls with normal liver function and structure). The pathologic criteria used to classify the alcoholic liver disease were: minimal lesions, steatosis, hepatitis without fibrosis, hepatitis with fibrosis and cirrhosis. The increments in the pressure values were directly related to the degree of structural liver damage. Intrahepatic manometry and liver biopsy, when performed together are useful parameters in the diagnosis of current anatomic and hemodynamic state of chronic alcoholic liver disease.

Measurement of intrahepatic pressure in alcoholic liver disease.

Intrahepatic pressure was measured in 172 subjects (148 patients with chronic alcoholic liver disease and 24 controls with normal liver function and structure). The pathologic criteria used to classify the alcoholic liver disease were: minimal lesions, steatosis, hepatitis without fibrosis, hepatitis with fibrosis and cirrhosis. The increments in the pressure values were directly related to the degree of structural liver damage. Intrahepatic manometry and liver biopsy, when performed together are useful parameters in the diagnosis of current anatomic and hemodynamic state of chronic alcoholic liver disease.