RESUMO
Although research has focused on the "innocence problem," "partial innocence" may also plague individuals who plead guilty to crimes they did not commit, but that are either comparable, more severe, or less severe than their actual crimes. Using a high-stake experimental paradigm and an immersive role-playing paradigm, we examined the psychology of partial innocence. Students were randomly induced (or imagined themselves) to be innocent, guilty, or partially innocent of committing an academic transgression and then given the choice to accept or reject a deal to avoid disciplinary sanction. Across three studies (Ns = 88, 75, 746), partially innocent students pled to cheating nearly as often as guilty students and vastly more often than innocent students. Partially innocent students-not unlike guilty students-experienced greater feelings of guilt than did innocent students. In turn, these feelings of guilt, but not shame, were associated with taking responsibility for a range of transgressions not committed.
RESUMO
BACKGROUND: Anabaptists comprise large and growing Amish and Mennonite populations with a unique genetic heritage and cultural background. Little is known about the symptoms and course of major mood disorders in Anabaptists. Even less is known about the impact of potential moderators on symptom severity and course. METHODS: A sample of Amish and Mennonite participants with bipolar, recurrent unipolar, or schizoaffective bipolar disorder (n = 155) were systematically evaluated with a well-validated instrument. Cases were compared with non-Anabaptist participants (n = 155) matched for age, sex, and psychiatric diagnosis and evaluated by the same methods. RESULTS: Despite substantial cultural differences, the profile of manic and depressive symptoms during illness episodes did not significantly differ between the two groups. Alcohol use disorder (AUD) was significantly less frequent among Anabaptists, and was associated with more major depressive episodes and more hospitalizations for major depression in Anabaptist, but not non-Anabaptist participants. Lifetime history of head injury showed a trend toward association with more episodes of major depression in both Anabaptist and non-Anabaptist groups that did not withstand multiple test correction. CONCLUSIONS: The presentation of a highly heritable psychiatric illness such as bipolar disorder does not differ in cases drawn from genetically unique Anabaptist populations. However, alcohol comorbidity, head injury, and their effects on illness course suggest some differences that deserve further investigation.
RESUMO
BACKGROUND: Neurocognitive deficits are present in bipolar disorder (BD) patients and their unaffected (nonbipolar) relatives, but it is not clear which domains are most often impaired and the extent of the impairment resulting from shared genetic factors. In this literature review, we address these issues and identify specific neurocognitive tasks most sensitive to cognitive deficits in patients and unaffected relatives. METHOD: We conducted a systematic review in Web of Science, PubMed/Medline and PsycINFO databases. RESULTS: Fifty-one articles assessing cognitive functioning in BD patients (23 studies) and unaffected relatives (28 studies) were examined. Patients and, less so, relatives show impairments in attention, processing speed, verbal learning/memory, and verbal fluency. CONCLUSION: Studies were more likely to find impairment in patients than relatives, suggesting that some neurocognitive deficits may be a result of the illness itself and/or its treatment. However, small sample sizes, differences among relatives studied (e.g., relatedness, diagnostic status, age), and differences in assessment instruments may contribute to inconsistencies in reported neurocognitive performance among relatives. Additional studies addressing these issues are needed.
