1.
Phys Rev B Condens Matter
; 47(4): 2251-2264, 1993 Jan 15.
Artigo
em Inglês
| MEDLINE
| ID: mdl-10006265
2.
Br J Surg
; 67(12): 890-2, 1980 Dec.
Artigo
em Inglês
| MEDLINE
| ID: mdl-7448518
RESUMO
The high incidence of gastroduodenal ulcers in patients with hyperparathyroidism has been ascribed to gastric hyperacidity induced by hypercalcaemia. In a clinical study comprising 40 patients with solitary parathyroid adenoma, a negative correlation between serum calcium and spontaneous gastric acid secretion was found. At the same time a positive correlation was found between serum calcium and gastrin. It is concluded that gastric hyperacidity is not part of the syndrome provoked by parathyroid adenomas, and that a mechanism inhibitory to gastrin is active in these patients. It is suggested that the inhibitor could be calcitonin.