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Exp Parasitol ; 116(4): 458-66, 2007 Aug.
Artigo em Inglês | MEDLINE | ID: mdl-17420014

RESUMO

Elimination of the helminth parasite Nippostrongylus brasiliensis from infected mice is mediated by IL-4 or IL-13 and dependent on the IL-4Ralpha chain and the transcription factor Stat6 in non-hematopoietic cells. However, it is not clear which Stat6-dependent effector molecules mediate worm expulsion. We identified intelectin-1 and -2 as Stat6-dependent genes that are induced during infection. Intelectins can bind galactofuranose, a sugar present only in microorganisms and might therefore serve as microbial pattern element. To analyze whether constitutive expression of intelectin-1 or -2 leads to accelerated pathogen clearance, transgenic mice were generated which express high levels of these genes selectively in the lung. Infection with N. brasiliensis or Mycobacterium tuberculosis did not result in accelerated pathogen clearance in transgenic as compared to wild-type mice. Further, no significant modulation of the immune response in lung or lymph nodes was observed. Thus, under these conditions, intelectins did not enhance pathogen clearance.


Assuntos
Citocinas/genética , Lectinas/genética , Nippostrongylus/genética , Fator de Transcrição STAT6/fisiologia , Infecções por Strongylida/genética , Animais , Citocinas/biossíntese , Citocinas/imunologia , Citometria de Fluxo , Proteínas Ligadas por GPI , Expressão Gênica , Mucosa Intestinal/metabolismo , Lectinas/biossíntese , Lectinas/imunologia , Pulmão/metabolismo , Camundongos , Camundongos Endogâmicos BALB C , Camundongos Transgênicos , Nippostrongylus/imunologia , Análise de Sequência com Séries de Oligonucleotídeos , Reação em Cadeia da Polimerase Via Transcriptase Reversa , Infecções por Strongylida/imunologia , Infecções por Strongylida/metabolismo
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