Dynamics and control of delayed rumor propagation through social networks.

Investigation of rumor spread dynamics and its control in social networking sites (SNS) has become important as it may cause some serious negative effects on our society. Here we aim to study the rumor spread mechanism and the influential factors using epidemic like model. We have divided the total population into three groups, namely, ignorant, spreader and aware. We have used delay differential equations to describe the dynamics of rumor spread process and studied the stability of the steady-state solutions using the threshold value of influence which is analogous to the basic reproduction number in disease model. Global stability of rumor prevailing state has been proved by using Lyapunov function. An optimal control system is set up using media awareness campaign to minimize the spreader population and the corresponding cost. Hopf bifurcation analyses with respect to time delay and the transmission rate of rumors are discussed here both analytically and numerically. Moreover, we have derived the stability region of the system corresponding to change of transmission rate and delay values.

Dynamics of COVID-19 transmission with comorbidity: a data driven modelling based approach.

An outbreak of the COVID-19 pandemic is a major public health disease as well as a challenging task to people with comorbidity worldwide. According to a report, comorbidity enhances the risk factors with complications of COVID-19. Here, we propose and explore a mathematical framework to study the transmission dynamics of COVID-19 with comorbidity. Within this framework, the model is calibrated by using new daily confirmed COVID-19 cases in India. The qualitative properties of the model and the stability of feasible equilibrium are studied. The model experiences the scenario of backward bifurcation by parameter regime accounting for progress in susceptibility to acquire infection by comorbidity individuals. The endemic equilibrium is asymptotically stable if recruitment of comorbidity becomes higher without acquiring the infection. Moreover, a larger backward bifurcation regime indicates the possibility of more infection in susceptible individuals. A dynamics in the mean fluctuation of the force of infection is investigated with different parameter regimes. A significant correlation is established between the force of infection and corresponding Shannon entropy under the same parameters, which provides evidence that infection reaches a significant proportion of the susceptible.