RESUMO
This report describes two infants with severe arterial hypertension secondary to unilateral renal artery stenosis which was manifested by polyuria, polydipsia, hypokalemic alkalosis, hyponatremia, increased natriuresis and increased plasma values of rennin and aldosterone. On sonographic examination, the contralateral non-stenotic kidney of both patients appeared enlarged and hyperechogenic mimicking parenchymal lesion. When the patients became normotensive, their sodium and potassium balance became normal and their contralateral non-stenotic kidney also became normal in size and echogenicity. The increase of the filtration and the natriuresis observed in the contralateral non-stenotic kidney of the patients with renovascular hypertension due to renal artery stenosis might be responsible for the hyperechogenicity. When the patients became normotensive, the filtration and excretion of sodium of the contralateral kidney also became normal and the increase of echogenicity also disappeared. The reversibility of the sonographic findings suggest a functional origin.
Assuntos
Alcalose/diagnóstico por imagem , Hipertensão Renovascular/diagnóstico por imagem , Hipopotassemia/diagnóstico por imagem , Rim/diagnóstico por imagem , Obstrução da Artéria Renal/diagnóstico por imagem , Alcalose/metabolismo , Alcalose/terapia , Pré-Escolar , Terapia Combinada , Humanos , Hipertensão Renovascular/metabolismo , Hipertensão Renovascular/terapia , Hipopotassemia/metabolismo , Hipopotassemia/terapia , Lactente , Masculino , Obstrução da Artéria Renal/metabolismo , Obstrução da Artéria Renal/terapia , UltrassonografiaAssuntos
Doenças do Ducto Colédoco/diagnóstico , Cistos/diagnóstico , Compostos de Organotecnécio , Criança , Pré-Escolar , Colangiografia , Feminino , Humanos , Iminoácidos , Lactente , Masculino , Pessoa de Meia-Idade , Compostos Organometálicos , Tomografia Computadorizada por Raios X , UltrassonografiaRESUMO
We describe a patient diagnosed of neuroblastoma IV-S who presented a hepatic calcification during the treatment. The different possibilities of hepatic calcification in the first year of life are commented.