1.
Neirofiziologiia
; 20(1): 32-7, 1988.
Artigo
em Russo
| MEDLINE
| ID: mdl-2454410
RESUMO
Interaction between anemone toxin BgTX8 and sodium channels in isolated single neurons of rat sensory ganglia was studied by voltage-clamp and intracellular perfusion techniques. It was shown that BgTX8 in external and internal solutions induced a dose-dependent slowdown of inactivation kinetics. Dissociation constant for receptor-toxin complex was found to be 4.10(-6) mol/l.