RESUMO
According to the authors' observations, the symptoms of nervous system derangement associated with legionnaires' disease rather often enter the disease structure and can virtually be characterized as a manifestation of infectious and toxic encephalopathy and polyneuropathy (encephalopolyneuropathy). In the majority of cases, the neurological disorders develop acutely or subacutely after or simultaneously with respiratory lesions. The clinical picture of encephalopathy is marked by permanent headache, mental abnormalities, memory disturbances, insomnia, pronounced astheno-vegetative and vascular manifestations. In patients with legionellosis, polyneuropathy is manifested by paresthesias, less frequently by pains in the distal parts of the limbs and myasthenia without visible atrophies. Vegetative disorders such as vegetative polyneuropathy of the hands and legs, visceral polyneuropathies are typical symptoms of the disease whatever its gravity. Vegetovascular dystonia together with long-term AP instability is an obligate sign of the disease. Electrophysiological examinations (EEG, REG, EMG) support the clinical findings and may serve the basis for an objective evaluation of the gravity of the neurological disorders. The degree of pulmonary lesions and the intensity of vegetative disorders eventually determine the torpidity and characteristics of the disease course.
Assuntos
Doença dos Legionários/complicações , Doenças do Sistema Nervoso/etiologia , Adulto , Eletroencefalografia , Eletromiografia , Feminino , Humanos , Doença dos Legionários/diagnóstico , Masculino , Pessoa de Meia-Idade , Doenças do Sistema Nervoso/diagnóstico , Polineuropatias/diagnóstico , Polineuropatias/etiologiaRESUMO
A rare syndrome is described. Combined investigation, including CT, yielded in a patient a typical clinical picture of nonatherosclerotic brain vessel calcification, especially within basal ganglia, parkinsonism, psychoemotional disorders, motor paroxysms. The syndrome etiology is discussed. Hypoparathyroidism with blood Ca metabolism and hereditary causes seem plausible. Differential diagnosis and treatment are described.
Assuntos
Calcinose/diagnóstico , Doenças Arteriais Cerebrais/diagnóstico , Calcinose/terapia , Doenças Arteriais Cerebrais/terapia , Terapia Combinada , Diagnóstico Diferencial , Humanos , Masculino , Pessoa de Meia-IdadeAssuntos
Herpes Zoster/diagnóstico , Adulto , Idoso , Feminino , Humanos , Masculino , Pessoa de Meia-Idade , RecidivaAssuntos
Herpes Zoster/complicações , Gânglio Trigeminal , Nervo Trigêmeo , Adolescente , Adulto , Idoso , Doenças dos Nervos Cranianos/complicações , Doenças dos Nervos Cranianos/tratamento farmacológico , Feminino , Herpes Zoster/tratamento farmacológico , Humanos , Masculino , Pessoa de Meia-IdadeRESUMO
The total bioelectrical activity of the muscles of the upper extremities as well as the neuromuscular transmission were examined in 30 patients with botulism at various stages of the disease. A picture of low-amplitude interference electromyogram was revealed. The results of stimulation electromyography pointed to a peculiar disorder of the neuromuscular transmission associated with a defect in the mediator release. Dynamic clinical and electromyographic observations pointed to a long period of the motor function restoration in convalescents.