Walleye Sander vitreus and smallmouth bass Micropterus dolomieu interactions: an historic stable-isotope analysis approach.

The carbon (δ(13) C) and nitrogen (δ(15) N) values of Sander vitreus scales differed pre and post-introduction in treatment lakes among years following the introduction of Micropterus dolomieu. No difference of δ(13) C and δ(15) N in S. vitreus scales was found in control lakes where M. dolomieu were not introduced. In treatment lakes, S. vitreus δ(15) N increased and δ(13) C decreased. No relationship was found between S. vitreus and M. dolomieu abundance in the two treatment lakes. Size structure of S. vitreus and M. dolomieu was negatively correlated and condition of the two species was positively correlated. Although S. vitreus feeding habits may have changed in the treatment lakes after M. dolomieu introductions, evidence suggests that fitness-related factors (i.e. abundance and condition) of S. vitreus remained unchanged, indicating S. vitreus and M. dolomieu may coexist where M. dolomieu have been introduced.

Autosomal dominant spinocerebellar ataxia with sensory axonal neuropathy (SCA4): clinical description and genetic localization to chromosome 16q22.1.

The hereditary ataxias represent a clinically and genetically heterogeneous group of neurodegenerative disorders. Various classification schemes based on clinical criteria are being replaced as molecular characterization of the ataxias proceeds; so far, seven distinct autosomal dominant hereditary ataxias have been genetically mapped in the human genome. We report linkage to chromosome 16q22.1 for one of these genes (SCA4) in a five-generation family with an autosomal dominant, late-onset spinocerebellar ataxia; the gene is tightly linked to the microsatellite marker D16S397 (LOD score = 5.93 at theta = .00). In addition, we present clinical and electrophysiological data regarding the distinct and previously unreported phenotype consisting of ataxia with the invariant presence of a prominent axonal sensory neuropathy.