Family Risk Exposure Profiles During Early Childhood: Developmental Processes and Adolescent Well-Being.

Although prior work indicates that exposure to multiple family risk factors negatively impacts adjustment in childhood and adolescence, few studies have examined whether children in high-risk families transition in and out of adversity during early childhood and whether patterns of change matter for adjustment in adolescence. Using data from a sample of 216 caregiver-child dyads participating in a study of prenatal cocaine exposure (116 exposed and 100 non-exposed; 50.9% girls), we used latent transition analysis to identify distinct profiles of early exposure to caregiver substance use (SU) and SU-related familial risk (caregiver psychological distress, exposure to violence, harshness, and low sensitivity) and the association between these profiles and adolescent well-being (i.e., hope, happiness, and life satisfaction). Assessments occurred when children were 13, 24, 36, and 48 months and during kindergarten (Mmonths = 66.16, SD = 4.47) and early adolescence (Myears = 13.26, SD = 0.88). Caregivers self-identified as 72.09% Black, 15.81% White, 10.23% Hispanic/Latinx, 1.40% other, and 0.47% American Indian. Four profiles of varying levels of exposure to caregiver SU and SU-related risks were identified from infancy to kindergarten: SU/family risks, no SU/low family risks, SU/negative parenting, and SU/low family risks. Most children stayed in the same profile (64.2%), while the rest transitioned between profiles. Children exposed to caregiver SU and family adversity had lower positive outcomes in adolescence. Stable membership in the SU/family risks profile had significant maladaptive consequences on adolescent well-being. Implications for research and the design of tailored interventions to promote well-being among at-risk youth are discussed.

Alcohol use during the COVID-19 pandemic: gender, parenthood, intimate partner violence, and stress.

Some preliminary work during the COVID-19 pandemic indicates that adult alcohol use increased, particularly for parents. This cross-sectional study examined the quantity and frequency of adults' alcohol use during the early stages of the pandemic. Additionally, the influences of gender, parenthood, COVID-19-related stressors and intimate partner violence (IPV) on alcohol consumption were examined. The sample consisted of 298 adults (98 parents) from across the United States who completed self-report surveys through Qualtrics at the beginning of the pandemic in May 2020. In the present study, all men reported higher levels of drinking compared to all women. Although stress levels did not impact alcohol consumption, findings indicate that increased IPV experiences were associated with higher levels of heavy drinking during the pandemic. Results also suggested that having children in the home particularly impacted drinking levels during the pandemic, above and beyond the influence of gender, IPV, and stress levels. These findings suggest that parenthood may have had a cascading influence on drinking experiences during the COVID-19 pandemic. Implications and recommendations for further research are discussed.

Prenatal substance exposure, early-life adversity, and parenting: Associations with adolescent stress response.

We tested a conceptual model examining associations between prenatal substance exposure and adolescent cortisol reactivity profiles in response to an acute social evaluative stressor. We included cortisol reactivity in infancy, and direct and interactive effects of early-life adversity and parenting behaviors (sensitivity, harshness) from infancy to early school age on adolescent cortisol reactivity profiles in model testing. Participants were 216 families (51% female children; 116 cocaine-exposed) recruited at birth, oversampled for prenatal substance exposure, and assessed from infancy to early adolescence (EA). Majority of participants self-identified as Black (72% mothers, 57.2% adolescents), and caregivers were primarily from low-income families (76%), were single (86%), and had high school or below education (70%) at recruitment. Latent profile analyses identified three cortisol reactivity patterns including elevated (20.4%), moderate (63.1%), and blunted (16.5%) reactivity groups. Prenatal tobacco exposure was associated with higher likelihood of membership in the elevated reactivity compared to the moderate reactivity group. Higher caregiver sensitivity in early life was associated with lower likelihood of membership in the elevated reactivity group. Prenatal cocaine exposure was associated with higher maternal harshness. Interaction effects among early-life adversity and parenting indicated that caregiver sensitivity buffered, and harshness exacerbated, the likelihood that high early adversity would be associated with the elevated and blunted reactivity groups. Results highlight the potential importance of prenatal alcohol and tobacco exposure for cortisol reactivity and the role of parenting as exacerbating or buffering the impact of early-life adversity on adolescent stress response.

Assessment of hostile intent attributions across escalating conflict stories.

Previous research suggests the importance of intent attributions in the development and maintenance of aggressive behavior. The primary purpose of the current study was to develop a measure assessing increases in attributions of hostility in response to escalating social conflict scenarios that were relational and instrumental in nature and to determine whether hostility trajectories were associated with relevant social experiences and behavior. A sample of primarily emerging adults (n = 750; M age = 19.97, SD = 3.60; 49.4% women, 48.3% men, 2.3% nonbinary or transgender; 69.9% Caucasian) responded to surveys regarding social behavior, peer victimization, and reports of hostile attribution biases in addition to the developed measure. Findings indicated that individuals adjusted their intent attributions across the conflict escalation stories, as reflected in linear increases in hostility ratings. Hostile attribution trajectories were also related to hostile attribution biases, peer victimization, and social behavior, including physical and relational aggression and prosocial behavior.

Applying a bifactor model to the functions of relational aggression: Associations with hostile attribution biases and difficulties with emotion regulation.

Previous research suggests that reactive and proactive aggression likely have distinct underlying mechanisms that uniquely contribute to the perpetration of each as a result of faulty cognitive and emotional processes. Still, very little work has examined the association of the functions of relational aggression with emotion dysregulation and hostile attribution biases. In addition, it is important to examine relational aggression in its pure and co-occurring functions given that past work finds reactive and proactive aggression to occur both jointly and distinctly. Thus, the current study employed a bifactor model to distinguish between pure reactive, pure proactive, and co-occurring relational aggression in emerging adulthood (N = 647, Mage = 19.92, SD = 2.83), a developmental time period for which relational aggression is particularly prominent. To further address gaps in the relational aggression literature, indirect pathway models revealed that relational hostile attribution biases emerged as a concurrent indirect path in the relationship between emotion dysregulation and pure reactive relational aggression. Furthermore, emotion dysregulation was directly positively associated with both pure functions as well as co-occurring relational aggression, and hostile attribution biases for relational provocations were directly associated with both pure functions of relational aggression, though the association was negative for pure proactive and positive for pure reactive relational aggression. Findings highlight the potential influence of emotion dysregulation and attributing hostile intent to social situations on engaging in the different functions of relational aggression.

The Indirect Effect of Peritraumatic Dissociation on the Relationship Between Childhood Maltreatment and Schizotypy.

Schizotypy is a multidimensional personality construct that is understood as a vulnerability for schizophrenia, often manifesting as more subtle and attenuated symptoms, referred to as schizotypic psychopathology. It has many well-established environmental risk factors, including experiencing childhood maltreatment (CM), but the intermediary mechanisms that relate CM to schizotypic psychopathology are unclear. Prior studies have demonstrated that trait dissociation may indirectly affect the relationship between CM and schizotypic psychopathology. However, less is known about the importance of peritraumatic dissociative experiences during CM and how it relates to schizotypic symptom manifestations in young adulthood. Therefore, the present study explored the independent contributions of peritraumatic and trait dissociation in the relationship between CM and schizotypy. Participants (N = 346) were undergraduate students who completed online self-report measures on CM, trait dissociation, peritraumatic dissociation experienced during CM, and schizotypic symptoms. The indirect effect of peritraumatic dissociation and trait dissociation on the relationship between CM and schizotypy was examined using mediational analyses. Correlational analyses revealed significant associations between self-reported CM, schizotypy, trait dissociation, and peritraumatic dissociation. In addition, mediational analyses indicated a significant indirect effect of peritraumatic dissociation (ß = .06, 95% confidence interval (CI) [0.01, 0.12]), but not trait dissociation (ß = .05, 95% CI [-0.02, 0.12]), on CM and schizotypy. These results highlight peritraumatic dissociation as an important mechanism driving the expression of schizotypic symptoms among individuals with a history of CM. Understanding how trauma sequelae lead to schizotypic psychopathology may be crucial in assessing and treating individuals with maltreatment histories or those on the psychosis spectrum.

Social and behavioral vulnerability, pregnancy, and negative mental health outcomes in the U.S. during the Covid-19 pandemic.

The novel coronavirus (Covid-19) pandemic has had a significant impact on the mental health of the general U.S. population. Extant literature has increasingly linked social vulnerabilities, risky behavior, and limited social and psychological resources to the growing mental health crisis during the virus's spread. The purpose of this study was to examine the impact of pertinent social vulnerabilities and subjective risk factors for both men and women on mental health (i.e., depression, anxiety, isolation) with a closer examination of the influence of pregnancy during the pandemic on mental health. The sample included 740,640 respondents participating in the U.S. Covid-19 Trends and Impact Survey that was deployed between February and March 2021. Descriptive statistics and ordinary least squares regression models are presented with a focus on the factors that shape negative mental health outcomes, particularly on the disparities between pregnant and non-pregnant women relative to men, but also subjective/perception factors (e.g., fear/worry) and social vulnerabilities. Results show that pregnant women were at significantly greater risk of negative mental health outcomes at this stage of the pandemic than either men or non-pregnant women. Overall, respondents who were younger, without children in the household, unemployed, worried generally about infection or their finances, or had ever tested positive for Covid were also more likely to report feelings of anxiety, isolation, and depression than their counterparts. Pregnant women may be especially vulnerable to depression, anxiety, and isolation during the pandemic and our findings suggest the importance of developing targeted mental health support for this sub-population.

Development of Bullying and Victimization: An Examination of Risk and Protective Factors in a High-Risk Sample.

This prospective longitudinal study from birth to late adolescence investigated how early risk predicted subsequent aggression in middle childhood and bullying perpetration, bullying victimization, and violence victimization in adolescence. In addition, the moderating role of protective factors (i.e., maternal sensitivity, positive peers, and school connectedness) on these associations were examined. Caregiver-infant dyads (N = 216; 72% Black/African American) were recruited as part of a longitudinal study on substance exposed youth. Data using multiple methods and informants (observations, interviews, caregiver, and child/youth self-reports) were collected from dyads in early childhood (EC, birth to 48 months), middle childhood (MC, i.e., 84 months), early adolescence (EA, M = 13.26 years, SD = .83) and later adolescence (LA, M = 15.08 years, SD = .83). A developmental cascading path model was tested. There were direct associations between EC maternal harsh parenting and aggression in MC. In turn, MC aggression was associated with higher violence victimization and bullying in EA. Finally, EA violence victimization was then associated with higher levels of bullying as well as victimization from bullying in LA. Consistent with predictions, there was also evidence that protective factors (i.e., maternal sensitivity and positive peers) moderated the impact of predictor variables on aggression and bullying outcomes. Specifically, maternal sensitivity moderated the link between EC and MC aggression, such that those with moderately high levels of maternal sensitivity showed a negative relation between EC and MC aggression, whereas those with low levels of maternal sensitivity showed continuity in aggression. Positive peer influence moderated the link between violence victimization in EA and bullying in LA, such that children high on both violence victimization and positive peers had the highest levels of bullying victimization.

Attachment as a Moderator in the Relation Between Child Maltreatment and Symptoms of Depression.

Experiencing child maltreatment is a risk factor for later psychopathology; however, not all survivors of child maltreatment go on to develop mental health diagnoses. There are likely important risk factors that act as moderators interacting with child maltreatment to contribute to the development of psychopathology. The present study examined the attachment dimensions of anxiety and avoidance as potential moderators in the association between child maltreatment and depressive symptomatology in a sample of college students. An attachment style high in anxiety or avoidance was expected to exacerbate the effect of child maltreatment on depressive symptomatology across both primary and secondary caregiver attachment relationships. This study was conducted at a private university in the northeastern United States in a sample of college students (N = 203; 52% male; Mage = 19.85, SDage = 2.19). Participants completed online measures of attachment, current mood symptoms, and demographic information. Two moderation models were tested, one for attachment to primary caregivers and one for attachment to secondary caregivers. Anxious attachment to primary caregivers moderated the relationship between child maltreatment and depressive symptoms (B = -0.16, p < .01, R2 =.44). However, moderation was not significant in the secondary caregiver attachment relationship. Maladaptive attachment styles, as well as child maltreatment itself, may result in disruptions in the development of positive internal working models of the self and others and adaptive emotion regulation. In cases of child maltreatment, interventions focused on the parent-child attachment relationship may have long-lasting effects and implications for the child's future mental health. This research highlights important areas of intervention in cases of child maltreatment as well as important differences in the anxiety and avoidance dimensions of attachment.

Child Community Violence Exposure in an at-Risk Sample: Developmental Trajectories, Caregiving Risks, and the Role of Child Temperament.

Objective: To better understand early etiological pathways to trajectories of child exposure to community violence (CECV), we used person-centered latent class growth analysis (LCGA) to examine chronicity of CECV from early school age through early adolescence, and examined early risks of the identified CECV trajectories (i.e., prenatal cocaine exposure, harsh parenting and caregiving instability across infancy and early childhood, and child activity level and inhibitory control at kindergarten age). Method: An at-risk sample (N = 216; 110 girls) of primarily low-income participants (76% on Temporary Assistance for Needy Families) with high rates of prenatal substance exposure was used. The majority of the mothers were African American (72%), had high school or below education (70%), and were single (86%). Postnatal assessments occurred at eight time points during infancy and toddlerhood, early childhood through early school age, and early adolescence. Results: We identified two distinct linearly increasing CECV trajectories (high-exposure and low-exposure). An interaction between child activity level and maternal harshness emerged, such that children with high activity levels and experiencing high harshness had the highest probabilities of being in the high exposure-increasing trajectory, in addition to early caregiving instability (conditional effect). Conclusion: The current findings not only have important theoretical implications but also provide insights into early intervention.

Developmental Pathways from Prenatal Substance Exposure to Reactive Aggression.

Few studies have examined etiological pathways from prenatal substance exposure to adolescent reactive aggression. We tested a conceptual model that included hypothesized pathways from prenatal substance exposure to adolescent aggression via autonomic reactivity and violence exposure from infancy to early school age and maternal harshness across early childhood. The sample included 216 families (106 boys) who primarily self-identified as Black or Mixed Race. Results supported the hypothesized path from violence exposure across early childhood and early school age to school age autonomic reactivity and early adolescent reactive aggression. There was also a significant interaction effect of sympathetic and parasympathetic reactivity on adolescent reactive aggression, with sympathetic arousal and parasympathetic suppression at early school age associated with higher reactive relational and physical aggression in adolescence. Results emphasize the importance of early experiences and autonomic nervous system changes in contributing to the cascade of risk for reactive aggression in early adolescence.

Prenatal exposure to opioids: Associations between the caregiving environment and externalizing behaviors.

Maternal opioid use during pregnancy is a rapidly growing public health crisis and is associated with a range of adverse developmental outcomes including externalizing behaviors among exposed children. Recent work has highlighted the role of indirect pathways from prenatal opioid exposure to behavioral outcomes through aspects of the caregiving environment, including parenting. This review highlights maternal sensitivity and related aspects of the caregiving environment that may impact the development of externalizing behaviors among children with a history of prenatal exposure to opioids. We conclude by providing suggestions for future directions in research examining development among children with prenatal opioid exposure.

Pathways From Early Family Violence to Adolescent Reactive Aggression and Violence Victimization.

PURPOSE: The current study examined how early childhood (EC) family violence and risk (i.e., maternal aggression, sibling aggression, environmental risk) predicted early adolescent (EA) reactive physical and relational aggression and violence victimization through middle childhood (MC) parenting (i.e., guilt induction, power assertive discipline). METHOD: Mother-infant dyads (N = 216; 72% African American) were recruited as part of a larger longitudinal study on prenatal cocaine and other substance exposure. Observations, interviews, and maternal and child self-report measures were collected from dyads in early childhood (1 to 36 months), middle childhood (84 months), and early adolescence (12 to 15 years). RESULTS: A cascading path model was specified where current variables were regressed on variables from the preceding time point. Primary results showed that environmental risk and EC child physical aggression predicted higher levels of MC caregiver power assertive discipline, which subsequently predicted lower levels of EA reactive relational aggression. Maternal substance use in pregnancy and the child's continuous placement with biological caregivers predicted higher levels of reactive physical aggression in EA. Finally, MC physical aggression and EA reactive relational aggression predicted higher levels of EA violence victimization. CONCLUSION: There were a series of direct paths from early childhood family violence and demographic factors to reactive aggression and violence victimization. The current study underscores the importance of evaluating multiple facets of family violence and risk when evaluating aggressive behavior and victimization.

Prenatal substance exposure and maternal hostility from pregnancy to toddlerhood: Associations with temperament profiles at 16 months of age.

We investigated whether infant temperament was predicted by level of and change in maternal hostility, a putative transdiagnostic vulnerability for psychopathology, substance use, and insensitive parenting. A sample of women (N = 247) who were primarily young, low-income, and had varying levels of substance use prenatally (69 nonsmokers, 81 tobacco-only smokers, and 97 tobacco and marijuana smokers) reported their hostility in the third trimester of pregnancy and at 2, 9, and 16 months postpartum, and their toddler's temperament and behavior problems at 16 months. Maternal hostility decreased from late pregnancy to 16 months postpartum. Relative to pregnant women who did not use substances, women who used both marijuana and tobacco prenatally reported higher levels of hostility while pregnant and exhibited less change in hostility over time. Toddlers who were exposed to higher levels of prenatal maternal hostility were more likely to be classified in temperament profiles that resemble either irritability or inhibition, identified via latent profile analysis. These two profiles were each associated with more behavior problems concurrently, though differed in their association with competence. Our results underscore the utility of transdiagnostic vulnerabilities in understanding the intergenerational transmission of psychopathology risk and are discussed in regards to the Research Domain Criteria (RDoC) framework.

Maternal Smoking and Psychosocial Functioning: Impact on Subsequent Breastfeeding Practices.

Introduction: Breastfeeding is a protective factor for women and children. Women who smoke cigarettes during pregnancy are less likely to initiate or persist in breastfeeding. However, less is known about why this is the case. Materials and Methods: The present study (n = 247) prospectively examined maternal/child factors that influence breastfeeding in a low-income, racially diverse at-risk sample of smoking and nonsmoking women. Pregnant women were recruited at their first prenatal appointment in an urban hospital and followed through 24-month postnatally. Women reported on the average number of cigarettes smoked/day during pregnancy, psychopathology, breastfeeding behavior, and infant reactivity. Results: Although a greater number of cigarettes smoked/day during pregnancy was associated with a lower likelihood of initiating or persisting in breastfeeding, maternal age, education, and infant reactivity offered predictive utility above and beyond maternal smoking. Conclusion: Smokers were less likely to initiate breastfeeding and breastfed for shorter duration than demographically similar nonsmokers; however, one of the mechanisms for reduced breastfeeding may be the psychosocial factors of younger age and lower education. Further, infant reactivity was also found to reduce the likelihood of initiating and persisting with breastfeeding.

Parent socialization of emotion in a high-risk sample.

The Socialization of Emotion (Eisenberg, Cumberland, & Spinrad, 1998; Eisenberg, Spinrad, & Cumberland, 1998) model creates a theoretical framework for understanding parents' direct and indirect influences on children's emotional development, including the influence of parent characteristics on subsequent emotion specific parenting. Large numbers of children live in families with fathers who have alcohol problems, setting the stage for cascading risk across development. For instance, fathers' alcohol problems are a marker of risk for higher family conflict, increased parental depression and antisociality, and less sensitive parenting, leading to dysregulated child emotion and behavior. We examined a conceptual model for emotion socialization in a community sample of alcoholic and nonalcoholic father families (N = 227) recruited in infancy (i.e., 12 months) with follow-ups to adolescence (i.e., 15-19 years), and examined if hypothesized paths differed by child sex or group status (alcoholic vs. nonalcoholic families). Results indicated significant indirect effects between parent psychopathology and sensitivity in early childhood to both adaptive (e.g., emotion regulation) and maladaptive (e.g., aggression and peer delinquency) outcomes in middle childhood to adolescence via child negative emotionality and supportive emotion socialization. There were significant differences by child sex and alcohol group status. Implications for intervention and prevention are discussed. (PsycINFO Database Record (c) 2020 APA, all rights reserved).

Parental influences on child report of relational attribution biases during early childhood.

Influences on social cognition, such as hostile attribution biases, are a relatively understudied topic despite the association of hostile attribution biases with important developmental and clinical outcomes. From a developmental perspective, it is particularly important to understand the early development of biases regarding how the intentions of others are perceived, especially in the relationship context. Therefore, understanding potential correlates, such as parental influences, of such biases during early childhood may be especially important. The current study (N = 121) was designed to explore several gaps and limitations in the hostile intent attribution literature, especially the lack of research on hostile attribution biases for relational provocations and associated parent behavior. In particular, this gap was addressed by investigating parental influences on hostile attribution biases for relational provocations during early childhood (i.e., 3-5 years of age) using parent and child reports. It was found that parent hostile attribution biases for relational provocations was significantly associated with child hostile attribution biases for relational provocations. Furthermore, it was demonstrated that parent relational aggression was related to parenting practices. Implications for research and practice are discussed.

A Longitudinal Study of Gun Violence Attitudes: Role of Childhood Aggression and Exposure to Violence, and Early Adolescent Bullying Perpetration and Victimization.

This prospective study examined the effects of early childhood physical aggression and violence exposure on bullying victimization/perpetration and attitudes toward guns and violence in early adolescence (EA) in a high-risk sample. Participants included 216 mother-child dyads from an ongoing longitudinal study using multi-method assessments (e.g., classroom observations, laboratory assessment, parent-, teacher-, and child self-reports). Results supported a developmental pathway from early adversity (i.e., prenatal substance use) to aggression at kindergarten age to bullying perpetration and gun violence attitudes (aggressive responses to shame) in EA. Higher peer victimization was also associated with aggressive responses to shame in EA. Results are discussed in light of the complexity of the motives for aggression and the need for prevention and early intervention.

Early Childhood Risk and Protective Factors Predicting Resilience against Adolescent Substance Use.

We examined associations between early childhood (first 3 years of life) risk and protective factors and resilience against adolescent substance use in a prospective sample of alcoholic and non-alcoholic families. We defined resilience as low or no substance use in the context of adversity (having a father with alcohol problems). The sample included 227 families recruited from birth records when children were 12 months old and followed longitudinally to 15-17 years of child ages (n = 182). Adolescents were grouped into 4 categories: Non-challenged (non-alcoholic parent, no adolescent substance use, n = 50), Troubled (non-alcoholic parent, adolescent substance use, n = 30), Resilient (alcoholic parent, no adolescent substance use, n = 36), and Vulnerable (alcoholic parent and adolescent substance use, n = 66). Multivariate analyses were used to examine group differences (resilient vs. vulnerable; non-challenged vs. troubled) in child and parent characteristics and family relationships domains. Children in the troubled group compared to non-challenged had lower effortful control and emotion-regulation, and those in the resilient group were more unadaptable or reactive to novelty compared to the vulnerable group. Parents of resilient compared to vulnerable children reported significantly lower alcohol symptoms and more partner aggression. Finally, fathers of resilient compared to vulnerable children were less aggravated with them in early childhood. Results highlight the importance of continuous measures of alcohol problems, early childhood functioning, and family characteristics for associations with adolescent risk and resilience.Passive gene-environment correlations may account for associations between parent alcohol problem severity and adolescent substance use.

Satter Eating Competence Inventory Subscale Restructure After Confirmatory Factor Analysis.

OBJECTIVE: To examine the structural validity of the empirically and theoretically derived Satter Eating Competence Inventory (ecSI 2.0). METHODS: Confirmatory factor analyses with 3 databases and a merger with 3 additional data sets (n = 2,010) from demographically heterogeneous samples. RESULTS: Findings supported retaining all 16 items and migration of the item I trust myself to eat enough for me from the Internal Regulation to Eating Attitudes subscales, providing 4 distinct but correlated subscales and an adequately fitting model, χ2(98) = 1,364.27, P < .001, comparative fit index = .91, root mean square error of approximation = .07, 95% confidence interval, 0.07-0.08, standardized root mean square residual = .05. CONCLUSION AND IMPLICATIONS: Psychometric integrity of the 16-item ecSI 2.0 was affirmed. Continued examination is recommended with cultural and linguistically diverse samples and network analyses to identify dynamic interactions among items.