Assuntos
Embolia Aérea/terapia , Oxigenoterapia Hiperbárica , Embolia e Trombose Intracraniana/terapia , Complicações na Gravidez/terapia , Comportamento Sexual , Adolescente , Embolia Aérea/etiologia , Embolia Aérea/fisiopatologia , Feminino , Humanos , Embolia e Trombose Intracraniana/etiologia , Embolia e Trombose Intracraniana/fisiopatologia , Gravidez , Complicações na Gravidez/etiologia , Complicações na Gravidez/fisiopatologiaAssuntos
Barotrauma/complicações , Mergulho/efeitos adversos , Pneumocefalia/etiologia , Adulto , Humanos , Espectroscopia de Ressonância Magnética , Masculino , Processo Mastoide/diagnóstico por imagem , Osso Petroso/lesões , Pneumocefalia/diagnóstico por imagem , Tomografia Computadorizada por Raios XRESUMO
The abundant blood supply normally found in the periorbital region grants the reconstructive surgeon many options for repair. When this blood supply is altered by such factors as thermal damage or scar formation, classic methods of lid reconstruction may not suffice. In such situations, treatment with hyperbaric oxygen accelerates the process of primary revascularization of full-thickness skin grafts and large composite grafts. Augmentation of capillary budding occurs because hyperbaric oxygen therapy raises the tissue oxygen tension in hypoxic areas to the level needed for extracellular deposition of collagen, which is needed for support of endothelial cells. Hyperbaric oxygen also appears to improve the survival of ischemic skin flaps of the face, although the exact mechanism of this action is unclear. Since 1982, a total of six patients needing periorbital reconstruction has been treated postoperatively with adjunct hyperbaric oxygen. Although the results have been uniformly favorable, a matched series comparing the results with and without hyperbaric oxygen therapy will be required to prove the efficacy of this treatment regimen.
Assuntos
Pálpebras/cirurgia , Face/cirurgia , Oxigenoterapia Hiperbárica , Cirurgia Plástica , Adolescente , Adulto , Feminino , Humanos , Masculino , Pessoa de Meia-Idade , Transplante de Pele , Retalhos Cirúrgicos , CicatrizaçãoRESUMO
The effect of acute carbon monoxide (CO) exposure on plasma adrenocorticotrophic hormone (ACTH), renin activity (PRA), aldosterone, and cortisol levels was studied in patients approximately 80-90 min after removal from the scene of exposure. Nine patients had carboxyhemoglobin (COHb) levels below 15%. Seven patients had COHb levels that exceeded 15% (Group II). Plasma ACTH, cortisol, and aldosterone levels were significantly higher in Group II (elevated COHb). Plasma cortisol and aldosterone levels were appropriate for the levels of ACTH and PRA achieved. We concluded that (a) acute carbon monoxide poisoning leads to elevated plasma corticosteroid levels, and (b) the adrenal gland appears to function normally acutely after exposure to carbon monoxide.
