[Effects of induction propofol mono-narcosis on the central and cerebral hemodynamics]. / Vliianie vvodnogo mononarkoza propofolom na tsentral'nuiu i tserebral'nuiu gemodinamiku.

Changes in the central hemodynamics were followed up by echocardiography and the linear velocity of the bloodflow by transcranial dopplerography in 15 patients with ASA classes I-II during induction bolus anesthesia with propofol in a dose 2.7 mg/kg without opioids. The diastolic and mean arterial pressure decreased by 12.3 and 15.4 %, respectively, the rate of cardiac contractions by 7.8%, cardiac index by 13.4%, and the resistive index on the radial and middle cerebral arteries by 13.4 and 10.2%, respectively, after administration of the above dose of propofol. The reaction to intubation of the trachea was the minimal, consisting in an increase of the mean and systolic arterial pressure by 22.2 and 22.7%, respectively, and of diastolic by 16.1%. The rest parameters were as initially. The linear velocity of the bloodflow in the middle cerebral artery did not reliably change. Hence, the central hemodynamic parameters are stable on condition that the deficit in the central bloodflow during induction anesthesia with propofol in a dose of 2.7 mg/kg is compensated for, adequate protection from intubation stress is provided, and autoregulation of the cerebral bloodflow is preserved.

[Artificial ventilation of the lungs in respiratory failure caused by lesions of the peripheral neuron]. / IVL pri dykhatel'noi nedostatochnosti, obuslovlennoi porazheniem perifericheskogo neirona.

A case with 155-day artificial ventilation of the lungs (AVL) in a patient with the poliencephalomyelitic form of tick-borne encephalitis is described. AVL was conducted through tracheostoma by the Puritan-Bennett 720ae device in the CMU mode for 26 days, then in the SIMU + PS accessory mode for 7 days, and in the CPAP + PS mode for 155 days. Despite the recovery of spontaneous respiration, vital capacity of the lungs did not normalize (1.1 liter) and the involvement at the level of the spine and the nuclei of the craniocerebral nerves was not corrected. The compensation was evidently due to training the diaphragm and the respiratory muscles and joining the accessory muscles.

[Clinical use of tramadol in lithotripsy compared with buprenorphine]. / Klinicheskoe ispol'zovanie tramadola pri litotripsii v sravnenii s buprenorfinom.

The comparison was made of analgetic action of tramadol and buprenorphine+diazepam in extracorporeal impulse lithotripsy. Opiate agonist-antagonist buprenorphine can provide high-quality analgesia in a drastic lowering of sHbO2. Nonnarcotic analgetic tramadol does not depress external respiration and provides adequate analgesia.