Authentic and hubristic pride: Differential effects on delay of gratification.

Research demonstrates that there are 2 distinct facets of pride: the prosocial, achievement-oriented form of pride known as authentic pride, and the self-aggrandizing, egotistical form of pride known as hubristic pride. This research examined whether authentic pride and hubristic pride have divergent effects on delay of gratification. Support was found for the prediction that authentic pride would facilitate the ability to delay gratification, whereas hubristic pride would undermine it. Also, self-transcendent value affirmation was demonstrated to moderate the effects of pride on delayed gratification. Specifically, when people feeling hubristic pride had an opportunity to affirm a self-transcendent value that was important to them, their tendency to seek immediate gratification was attenuated. Theoretical implications of these findings are discussed. (PsycINFO Database Record

Genetic studies of Prader-Willi patients provide evidence for conservation of genomic architecture in proximal chromosome 15q.

Prader-Willi syndrome (PWS) is a neurogenetic disorder associated with recurrent genomic recombination involving low copy repeats (LCRs) located in the human chromosome 15q11-q13. Previous studies of PWS patients from Asia suggested that there is a higher incidence of deletion and lower incidence of maternal uniparental disomy (mUPD) compared to that of Western populations. In this report, we present genetic etiology of 28 PWS patients from Taiwan. Consistent with the genetic etiology findings from Western populations, the type II deletion appears to be the most common deletion subtype. Furthermore, the ratio of the two most common deletion subtypes and the ratio of the maternal heterodisomy to isodisomy cases observed from this study are in agreement with previous findings from Western populations. In addition, we identified and further mapped the deletion breakpoints in two patients with atypical deletions using array CGH (comparative genomic hybridization). Despite the relatively small numbers of patients in each subgroup, our findings suggest that the genomic architecture responsible for the recurrent recombination in PWS is conserved in Taiwanese of the Han Chinese heritage and Western populations, thereby predisposing chromosome 15q11-q13 to a similar risk of rearrangements.