RESUMO
GeSn is a group IV alloy material with a narrow bandgap, making it favorable for applications in sensing and imaging. However, strong surface carrier recombination is a limiting factor. To overcome this, we investigate the broadband photoelectrical properties of graphene integrated with doped GeSn, from the visible to the near infrared. It is found that photo-generated carriers can be separated and transported with a higher efficiency by the introduction of the graphene layer. Considering two contrasting arrangements of graphene on p-type and n-type GeSn films, photocurrents were suppressed in graphene/p-type GeSn heterostructures but enhanced in graphene/n-type GeSn heterostructures when compared with control samples without graphene. Moreover, the enhancement (suppression) factor increases with excitation wavelength but decreases with laser power. An enhancement factor of 4 is achieved for an excitation wavelength of 1064 nm. Compared with previous studies, it is found that our graphene/n-type GeSn based photodetectors provide a much wider photodetection range, from 532 nm to 1832 nm, and maintain comparable responsivity. Our experimental findings highlight the importance of the induced bending profile on the charge separation and provides a way to design high performance broadband photodetectors.
RESUMO
The effects of a plastic objective lens's astigmatism on the push-pull tracking-error signal (TES) of an optical disk data storage system were investigated theoretically and experimentally. Astigmatism of plastic objective lenses arises commonly from the asymmetric deviation from their designed shape during the molding process. By carefully studying the aberration characteristics of the objective lens and including the astigmatism of the laser diode in the analysis, we can calculate the combined effects of astigmatism of these two components on the push-pull TES. It is found, from both the simulations and the experiments, that, by rotation of the objective lens about the optical axis, the peak-to-peak value of the push-pull TES varies with the lens's rotation angle, and a change as great as 340% in its value was observed in a given optical pickup.
RESUMO
One can control the aberrations of an optical readout system by varying the width of a strip of antireflection coatings deposited upon plastic objective lenses. It is found that one can control the magnitude of the third-order astigmatism of the system by changing the coating width. This process has the advantage that it does not significantly cause other kinds of aberration such as coma and spherical aberrations to deteriorate. When these nonrotational symmetrically (NRS) coated lenses are used for off-axis operations such as tracking movements in optical drives, the change in the magnitude of the astigmatism (DAS) that is generated can be made smaller than those of symmetrically coated or noncoated lenses. As much as a 73% decrease in DAS was observed experimentally with a NRS-coated lens. Including the birefringence of the plastic material in the analysis yields a low and constant level of astigmatism generated by shifting of the objective lens.
RESUMO
Histamine and serotonin levels in gastric secretion and the effects of pharmacological antagonists were studied in rats in which stomach ulceration was induced by electrical vagal stimulation. Electrical vagal stimulation (2 and 5 V) produced a graded increase in haemorrhagic glandular mucosal ulcers. NaHCO3 perfusion completely neutralised the increased acid output but failed to prevent ulceration. Atropine inhibited gastric mast cell degranulation as well as histamine and serotonin release. Diphenhydramine, atropine and sub-diaphragmatic vagotomy antagonised the increase in intragastric pressure. Diphenhydramine, cimetidine, atropine or vagotomy but not methysergide reduced ulcer severity. It is concluded that gastric acid and serotonin do not play an important role in glandular ulceration induced by electrical vagal stimulation. The lesions probably result from increased intragastric pressure and release of gastric histamine which stimulates H1 and H2 receptors in the stomach. The similarities between the aetiologies of glandular ulcers due to electrical vagal stimulation and to stress are also discussed.
