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1.
Actas dermo-sifiliogr. (Ed. impr.) ; 114(2): 125-131, feb. 2023. ilus, tab
Artigo em Espanhol | IBECS | ID: ibc-215416

RESUMO

El consumo de cocaína, junto con algunos de sus adulterantes más frecuentes como el levamisol, puede provocar múltiples procesos cutáneos y mucosos, ya sean de índole isquémico, dermatosis neutrofílicas, lesiones destructivas de la línea media y vasculitis asociadas a ANCA, entre otros. Generalmente no se asocia clínica sistémica llamativa.Todos estos cuadros pueden presentar anticuerpos antinucleares, antifosfolípido y contra distintos antígenos de los neutrófilos, en ocasiones con un patrón característico. El estudio histológico suele mostrar cambios vasculares como vasculitis leucocitoclástica, necrosis de la pared y trombos. En este artículo revisamos las características clínicas, serológicas e histológicas de estas entidades, junto con los mecanismos fisiopatológicos implicados, el diagnóstico diferencial y su tratamiento. (AU)


Cocaine and some of its main adulterants, such as levamisole, can cause multiple cutaneous and mucosal manifestations, including ischemic complications, neutrophilic dermatoses, midline destructive lesions, and vasculitis associated with antineutrophil cytoplasmic antibodies (ANCAs). Striking systemic symptoms are generally not seen.In all these conditions, positive test results may be observed for antinuclear antibodies, antiphospholipid antibodies, and various ANCAs, sometimes with characteristic staining patterns. Histology typically shows vascular changes, such as leukocytoclastic vasculitis, necrotizing vasculitis, and thrombi. We review the clinical, serologic, and histologic features of cutaneous and mucosal conditions associated with the use of cocaine and also look at pathophysiologic mechanisms, differential diagnoses, and treatments. (AU)


Assuntos
Humanos , Transtornos Relacionados ao Uso de Cocaína/complicações , Dermatopatias/etiologia , Cocaína/efeitos adversos , Levamisol/efeitos adversos , Vasculite Leucocitoclástica Cutânea/diagnóstico , Vasculite Leucocitoclástica Cutânea/etiologia , Diagnóstico Diferencial , Transtornos Relacionados ao Uso de Cocaína/diagnóstico , Transtornos Relacionados ao Uso de Cocaína/fisiopatologia , Pele/efeitos dos fármacos , Pele/patologia , Dermatopatias/diagnóstico , Dermatopatias/fisiopatologia
2.
Actas dermo-sifiliogr. (Ed. impr.) ; 114(2): t125-t131, feb. 2023. ilus, tab
Artigo em Inglês | IBECS | ID: ibc-215417

RESUMO

Cocaine and some of its main adulterants, such as levamisole, can cause multiple cutaneous and mucosal manifestations, including ischemic complications, neutrophilic dermatoses, midline destructive lesions, and vasculitis associated with antineutrophil cytoplasmic antibodies (ANCAs). Striking systemic symptoms are generally not seen. In all these conditions, positive test results may be observed for antinuclear antibodies, antiphospholipid antibodies, and various ANCAs, sometimes with characteristic staining patterns. Histology typically shows vascular changes, such as leukocytoclastic vasculitis, necrotizing vasculitis, and thrombi. We review the clinical, serologic, and histologic features of cutaneous and mucosal conditions associated with the use of cocaine and also look at pathophysiologic mechanisms, differential diagnoses, and treatments. (AU)


El consumo de cocaína, junto con algunos de sus adulterantes más frecuentes como el levamisol, puede provocar múltiples procesos cutáneos y mucosos, ya sean de índole isquémico, dermatosis neutrofílicas, lesiones destructivas de la línea media y vasculitis asociadas a ANCA, entre otros. Generalmente no se asocia clínica sistémica llamativa. Todos estos cuadros pueden presentar anticuerpos antinucleares, antifosfolípido y contra distintos antígenos de los neutrófilos, en ocasiones con un patrón característico. El estudio histológico suele mostrar cambios vasculares, como vasculitis leucocitoclástica, necrosis de la pared y trombos. En este artículo revisamos las características clínicas, serológicas e histológicas de estas entidades, junto con los mecanismos fisiopatológicos implicados, el diagnóstico diferencial y su tratamiento. (AU)


Assuntos
Humanos , Transtornos Relacionados ao Uso de Cocaína/complicações , Dermatopatias/etiologia , Cocaína/efeitos adversos , Levamisol/efeitos adversos , Vasculite Leucocitoclástica Cutânea/diagnóstico , Vasculite Leucocitoclástica Cutânea/etiologia , Diagnóstico Diferencial , Transtornos Relacionados ao Uso de Cocaína/diagnóstico , Transtornos Relacionados ao Uso de Cocaína/fisiopatologia , Pele/efeitos dos fármacos , Pele/patologia , Dermatopatias/diagnóstico , Dermatopatias/fisiopatologia
3.
Actas Dermosifiliogr ; 114(2): 125-131, 2023 Feb.
Artigo em Inglês, Espanhol | MEDLINE | ID: mdl-36115385

RESUMO

Cocaine and some of its main adulterants, such as levamisole, can cause multiple cutaneous and mucosal manifestations, including ischemic complications, neutrophilic dermatoses, midline destructive lesions, and vasculitis associated with antineutrophil cytoplasmic antibodies (ANCAs). Striking systemic symptoms are generally not seen. In all these conditions, positive test results may be observed for antinuclear antibodies, antiphospholipid antibodies, and various ANCAs, sometimes with characteristic staining patterns. Histology typically shows vascular changes, such as leukocytoclastic vasculitis, necrotizing vasculitis, and thrombi. We review the clinical, serologic, and histologic features of cutaneous and mucosal conditions associated with the use of cocaine and also look at pathophysiologic mechanisms, differential diagnoses, and treatments.


Assuntos
Transtornos Relacionados ao Uso de Cocaína , Cocaína , Vasculite Leucocitoclástica Cutânea , Vasculite , Humanos , Pele/patologia , Transtornos Relacionados ao Uso de Cocaína/complicações , Transtornos Relacionados ao Uso de Cocaína/diagnóstico , Transtornos Relacionados ao Uso de Cocaína/patologia , Vasculite Leucocitoclástica Cutânea/complicações , Vasculite Leucocitoclástica Cutânea/patologia , Cocaína/efeitos adversos , Levamisol/efeitos adversos , Anticorpos Anticitoplasma de Neutrófilos
8.
Actas dermo-sifiliogr. (Ed. impr.) ; 99(3): 173-184, abr. 2008. tab
Artigo em Es | IBECS | ID: ibc-62821

RESUMO

El hábito de fumar constituye la principal causa evitable de enfermedad y muerte en el mundo occidental. Su consumo se relaciona directamente con enfermedades cardiovasculares, bronquitis crónica y numerosos procesos neoplásicos. El tabaco ejerce asimismo múltiples y diversos efectos sobre la piel, la mayor parte de ellos nocivos. En este sentido, el hábito de fumar se halla estrechamente relacionado con numerosas enfermedades dermatológicas como la psoriasis, la pustulosis palmoplantar, la hidrosadenitis supurativa, el lupus eritematoso sistémico y discoide, así como con procesos neoplásicos del labio, de la cavidad oral y de la región anogenital, entre otros. Existe una relación controvertida con el melanoma, el carcinoma escamoso de la piel, el carcinoma basocelular y el acné. Por otra parte, este hábito parece que ejercería un efecto protector frente al desarrollo de las aftas orales, de la rosácea, del herpes simple labial, del pénfigo vulgar y de la dermatitis herpetiforme. Aparte de la influencia que el hábito de fumar ejerce sobre las enfermedades dermatológicas, el consumo de tabaco es también responsable directo de distintas dermatosis como la estomatitis nicotínica, la lengua negra vellosa, la enfermedad periodontal, así como de algunos tipos de urticaria y de dermatitis de contacto. Además, no hay que olvidar la repercusión cosmética de este hábito, que conlleva una pigmentación amarilla de los dedos y de las uñas, una alteración del color normal de los dientes, una disminución del gusto y del olfato, halitosis e hipersalivación y un desarrollo precoz de arrugas faciales (AU)


Smoking is the main modifiable cause of disease and death in the developed world. Tobacco consumption is directly linked to cardiovascular disease, chronic bronchitis, and many malignant diseases. Tobacco also has many cutaneous effects, most of which are harmful. Smoking is closely associated with several dermatologic diseases such as psoriasis, pustulosis palmoplantaris, hidrosadenitis suppurativa, and systemic and discoid lupus erythematosus, as well as cancers such as those of the lip, oral cavity, and anogenital region. A more debatable relationship exists with melanoma, squamous cell carcinoma of the skin, basal cell carcinoma, and acne. In contrast, smoking seems to protect against mouth sores, rosacea, labial herpes simplex, pemphigus vulgaris, and dermatitis herpetiformis. In addition to the influence of smoking on dermatologic diseases, tobacco consumption is also directly responsible for certain dermatoses such as nicotine stomatitis, black hairy tongue, periodontal disease, and some types of urticaria and contact dermatitis. Furthermore, we should not forget that smoking has cosmetic repercussions such as yellow fingers and fingernails, changes in tooth color, taste and smell disorders, halitosis and hypersalivation, and early development of facial wrinkles (AU)


Assuntos
Humanos , Masculino , Feminino , Fatores de Risco , Tabagismo/efeitos adversos , Dermatopatias Papuloescamosas/complicações , Dermatopatias Eczematosas/complicações , Envelhecimento da Pele , Envelhecimento da Pele/fisiologia , Dermatite/complicações , Neoplasias Cutâneas/complicações , Dermatopatias/complicações , Dermatopatias/diagnóstico , Poluição por Fumaça de Tabaco/efeitos adversos , Psoríase/complicações , Hidradenite/complicações , Lúpus Eritematoso Sistêmico/complicações , Estomatite/complicações , Pênfigo/complicações , Carcinoma Basoescamoso/complicações , Urticária/complicações , Dermatopatias/epidemiologia
9.
Actas Dermosifiliogr ; 99(3): 173-84, 2008 Apr.
Artigo em Espanhol | MEDLINE | ID: mdl-18358192

RESUMO

Smoking is the main modifiable cause of disease and death in the developed world. Tobacco consumption is directly linked to cardiovascular disease, chronic bronchitis, and many malignant diseases. Tobacco also has many cutaneous effects, most of which are harmful. Smoking is closely associated with several dermatologic diseases such as psoriasis, pustulosis palmoplantaris, hidrosadenitis suppurativa, and systemic and discoid lupus erythematosus, as well as cancers such as those of the lip, oral cavity, and anogenital region. A more debatable relationship exists with melanoma, squamous cell carcinoma of the skin, basal cell carcinoma, and acne. In contrast, smoking seems to protect against mouth sores, rosacea, labial herpes simplex, pemphigus vulgaris, and dermatitis herpetiformis. In addition to the influence of smoking on dermatologic diseases, tobacco consumption is also directly responsible for certain dermatoses such as nicotine stomatitis, black hairy tongue, periodontal disease, and some types of urticaria and contact dermatitis. Furthermore, we should not forget that smoking has cosmetic repercussions such as yellow fingers and fingernails, changes in tooth color, taste and smell disorders, halitosis and hypersalivation, and early development of facial wrinkles.


Assuntos
Dermatopatias/etiologia , Fumar/efeitos adversos , Humanos , Envelhecimento da Pele , Neoplasias Cutâneas/etiologia
10.
Rev Clin Esp ; 198(2): 95-8, 1998 Feb.
Artigo em Espanhol | MEDLINE | ID: mdl-9558924

RESUMO

The aggressive variant of the classical Kaposi sarcoma (KS), though uncommon, has an utmost relevance because its evolutive characteristics entail a great morbidity and mortality. Three classical KS cases are reported which manifested an unusually aggressive course, with extensive cutaneous and visceral involvement. One of the patients was diagnosed of a non-Hodgkin lymphoma 12 months later. None of the three patients responded to the different therapeutic regimens used: polychemotherapy, monochemotherapy and interferon-alfa, respectively. The three patients died within two years after diagnosis. In KS patients with pulmonary involvement, working in coal mines was recorded as occupational antecedent. These three cases illustrate the different clinical and therapeutic characteristics of an uncommon subtype in the KS spectrum in general and of its classical variant in particular. Likewise, the possible role of occupational exposure in the etiopathogenesis of KS is discussed.


Assuntos
Sarcoma de Kaposi/fisiopatologia , Idoso , Humanos , Linfoma não Hodgkin/complicações , Masculino , Pessoa de Meia-Idade , Exposição Ocupacional , Sarcoma de Kaposi/complicações , Sarcoma de Kaposi/etiologia
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