[Mechanism of eclamptic coma development].

Analysis of the course of the disease in 28 puerperas with eclamptic coma has ascertained that it develops only in the presence of hypokinetic circulation as a result of lower circulating blood volume and cardiac index, which is followed by a cerebral circulation reduction to the critical values due to blood flow in the cortical structures. To maintain high blood flow in the subcortical structures is one of the leading causes of vasogenic edema of brain glia, followed by impaired liquorodynamics, which indices eclampsia. Low sustained cerebral circulation as a consequence of inadequate intensive care favors the extent of edema to the cerebral cortical structures after relieving eclampsia.

[Effect of dipyridamole on blood levels of fibroblast growth factor and vascular endothelial growth factor in patients with ischemic heart disease]. / Vliianie dipiridamola na soderzhanie v krovi sosudistogo éndotelial'nogo faktora rosta i faktora rosta fibroblastov u bol'nykh ishemicheskoi bolezn'iu serdtsa.

Addition if dipyridamole to standard therapy in patients with ischemic heart disease was associated with improvement of functional class of angina and elevation of blood levels of fibroblast growth factor and Vascular Endothelial Growth Factor.