Tsunamis generated by eruptions from mount st. Augustine volcano, alaska.

During an eruption of the Alaskan volcano Mount St. Augustine in the spring of 1986, there was concern about the possibility that a tsunami might be generated by the collapse of a portion of the volcano into the shallow water of Cook Inlet. A similar edifice collapse of the volcano and ensuing sea wave occurred during an eruption in 1883. Other sea waves resulting in great loss of life and property have been generated by the eruption of coastal volcanos around the world. Although Mount St. Augustine remained intact during this eruptive cycle, a possible recurrence of the 1883 events spurred a numerical simulation of the 1883 sea wave. This simulation, which yielded a forecast of potential wave heights and travel times, was based on a method that could be applied generally to other coastal volcanos.

[Liver diseases with microsomal antibodies. Frequency, clinical and immunological findings and course observations (author's transl)]. / Lebererkrankungen mit mikrosomalen Antikörpern Häufigkeit, klinische und immunologische Befunde sowie Verlaufsbeobachtungen.

Microsomal antibodies (LKM-antibodies) differ in the indirect immunfluorescence by use of rat organ sections (kidney, stomach, and liver) from mitochondrial antibodies by there reaction with proximal renal tubules and hepatocytes while parietal cells usually fail to stain. In contrast to other humoral autoantibodies LKM-antibodies are rare. In a prospective study on 930 adults and 33 children with two third chronic hepatitis resp. cirrhosis frequency in the first group amounts to 0.38% and in the latter to 6.9% of all liver diseases. Among six patients with LKW-antibodies and chronic active hepatitis there were three children aged 4-13 and three adults between 45-55 years. On common results two children showed beside LKM-antibodies by absence of other immunphenomenons a rise of gamma-globulins in the electrophoresis and immunglobulin G as well as a chronic active hepatitis with necrosis leading in spite of initial rapid progression and persistent antibodies into inactive postnecrotiv cirrhosis. The other four patients showed no common course in respect to clinical, histological and immunological findings.

[Frequency of chronic non-suppurative destructive cholangitis and primary biliary cirrhosis in comparison with other chronic inflammatory liver diseases]. / Häufigkeit der chronischen nichteitrigen destruierenden Cholangitis und primär biliären Leberzirrhose im Vergleich mit anderen chronischen entzündlichen Lebererkrankungen

Primary biliary liver disease was diagnosed and verified by histology and demonstration of antibodies against mitochondrial proteins in 73 patients between 1970 and 1974. Comparing cases between 1950 and 1961 and between 1970 and 1974 with the frequency of other chronic inflammatory liver diseases it was found that figures for advanced primary biliary cirrhosis remained unchanged during both periods whereas early stages of primary biliary disease were detected far more frequently in the latter period. One case with chronic non-suppurative destructive cholangitis or complete primary biliary cirrhosis was outnumbered by 8 cases of chronic aggressive hepatitis and 15 of liver cirrhosis of various aetiologies in Kassel. The relations in Göttingen were 1 : 5 and 1 : 6, respectively. Primary biliary diseases are no longer rare.

Microearthquakes at st. Augustine volcano, alaska, triggered by Earth tides.

Microearthquake activity at St. Augustine volcano, located at the mouth of Cook Inlet in the Aleutian Islands, has been monitored since August 1970. Both before and after minor eruptive activity on 7 October 1971, numerous shallow-foci microearthquake swarms were recorded. Plots of the hourly frequency of microearthquakes often show a diurnal peaking of activity. A cross correlation of this activity with the calculated magnitudes of tidal acceleration exhibited two prominent phase relationships. The first, and slightly more predominant, phase condition is a phase delay in the microearthquake activity of approximately 1 hour from the time of maximum tidal acceleration. This is thought to be a direct microearthquake-triggering effect caused by tidal stresses. The second is a phase delay in the microearthquake activity of approximately 5 hours, which correlates well with the time of maximum oceanic tidal loading. Correlation of the individual peaks of swarm activity with defined components of the tides suggests that it may be necessary for tidal stressing to have a preferential orientation in order to be an effective trigger of microearthquakes.