RESUMO
We study the phase diagram and quantum critical region of one of the fundamental models for electronic correlations: the periodic Anderson model. Employing the recently developed dynamical vertex approximation, we find a phase transition between a zero-temperature antiferromagnetic insulator and a Kondo insulator. In the quantum critical region, we determine a critical exponent γ=2 for the antiferromagnetic susceptibility. At higher temperatures, we have free spins with γ=1 instead, whereas at lower temperatures, there is an even stronger increase and suppression of the susceptibility below and above the quantum critical point, respectively.
RESUMO
Arteriosclerosis obliterans (ASO) has become one of major health problems in the elderly people in Japan. This paper reviews the recent progress in the diagnosis and treatment of ASO. Both medical and surgical treatments, such as atherectomy catheters, endovascular metallic stenting, laser angioplasty and LDL-apheresis have recently been greatly developed. As atherosclerosis is a generalised disease, patients with ASO tend to have other atherosclerotic diseases, i.e., cerebrovascular disease, coronary heart disease and renal vascular disease. For the selection of treatments, we have to consider the whole background of the patients, including a quality of life.
Assuntos
Arteriosclerose Obliterante/diagnóstico , Angioplastia a Laser , Arteriosclerose Obliterante/terapia , Aterectomia , Determinação da Pressão Arterial , Humanos , Stents , TermografiaRESUMO
A further case of ring chromosome 15 in a 12-year-old boy with growth failure is described. He had minor congenital anomalies, but almost normal intelligence.
Assuntos
Aberrações Cromossômicas , Cromossomos Humanos Par 15 , Transtornos do Crescimento/genética , Cromossomos em Anel , Criança , Bandeamento Cromossômico , Humanos , Inteligência , Cariotipagem , MasculinoRESUMO
A case of hereditary spherocytosis (HS) is reported. Cytogenetic study revealed a de novo minute deletion of chromosome 8. The critical portion which affected the expression of the HS phenotype appeared to be localized to 8p11.22----8p21.1.