RESUMO
Poor nutrition and obesity can directly lead to pathological conditions managed by physical therapists or negatively influence recovery from movement dysfunction. The physical therapist/client relationship provides an opportunity for screening for poor nutrition as well as recommending and supporting better nutrition practices by the clients under their care. As such, it is important for the physical therapy professional to understand optimal nutrition for healthy living and serve as a consultant for better nutrition for their clients. To achieve this end, this article addresses strategies for identifying nutritional trends for the specific groups of clients, screening for nutritional problems, assessing clients' readiness to change eating habits, providing useful information and resources concerning optimal nutrition, and recognizing the need for referral to nutrition professionals.
Assuntos
Dieta/efeitos adversos , Medicina Baseada em Evidências , Distúrbios Nutricionais/dietoterapia , Modalidades de Fisioterapia , Redução de Peso , Aconselhamento , Comportamento Alimentar , Comportamentos Relacionados com a Saúde , Conhecimentos, Atitudes e Prática em Saúde , Humanos , Desnutrição/dietoterapia , Desnutrição/etiologia , Desnutrição/fisiopatologia , Distúrbios Nutricionais/diagnóstico , Distúrbios Nutricionais/etiologia , Distúrbios Nutricionais/fisiopatologia , Obesidade/dietoterapia , Obesidade/etiologia , Obesidade/fisiopatologia , Sobrepeso/dietoterapia , Sobrepeso/etiologia , Sobrepeso/fisiopatologia , Educação de Pacientes como Assunto , Especialidade de Fisioterapia , Relações Profissional-Paciente , Encaminhamento e Consulta , Comportamento de Redução do Risco , Resultado do TratamentoRESUMO
We evaluated whether a daily high-dose calcium supplement perturbs the zinc status in 23 postmenopausal women (mean age: 63 yr) with low bone mineral density. Plasma and erythrocyte zinc concentrations, plasma bone-specific alkaline phosphatase (BSAP) and 5'-nucleotidase activities, and urinary zinc and calcium excretion were determined first at the end of 4 wk of daily oral calcium (1200 mg) and were measured again at the end of the subsequent 4 wk of daily cosupplementation with calcium (1200 mg) and zinc (30 mg). Mean plasma and erythrocyte zinc concentrations after 4 wk of calcium alone were not significantly different from concentrations after cosupplementation of calcium and zinc. Mean plasma BSAP activities before cosupplementation with zinc was significantly higher than that after zinc (p < 0.02), whereas plasma 5'-nucleotidase activities were not affected by zinc supplementation. Urinary zinc excretion slightly, but significantly, increased after the supplementation of zinc (p < 0.05), whereas calcium excretion remained similar. Our data indicate that a 4-wk zinc supplementation did not significantly improve zinc status. Although limited by the small sample size and short study duration, our data suggest that a daily calcium dose of 1200 mg had no effect on the zinc status of our subjects.