Fabrication of Ni-Polyaniline/Graphene Oxide Composite Electrode with High Capacitance and Water Splitting Activity.

The Ni-PANI@GO composite electrode was fabricated via cost effective electrodeposition technique. According to the XRD, FTIR, Raman, SEM, and XPS analyses revealed that the nickel doped PANI@GO composite has been fabricated on the surface of the nickel foam. Addition of nickel significantly enhanced interaction between graphene with PANI leading to higher degree of polyaniline doping though imine groups. Electrochemical investigation revelated the significant performance of the Ni-PANI@GO composite electrode, boosting an impressive capacitance of 4480 F/g at 40 A/g, surpassing previous Ni-foam-based binder-free electrodes. Notably, Ni-PANI@GO electrode displayed excellent catalytic activity in both oxygen evolution reaction (OER) and hydrogen evolution reaction (HER), generating a considerable volume of the gas bubbles at relatively modest overpotentials of 279 mV and 244 mV respectively. This event allows for the achievement of 20 mA cm-2 current density. Furthermore, in the laboratory-scale water electrolyzer, a low cell voltage of 1.72 V was achieved, facilitating a water-splitting current density of 20 mA cm-2. This study underscores the premising potential for the real-world device's application of the versatile Ni-PANI@GO composite electrode.

Proliferative Funiculitis-Like Dedifferentiated Liposarcoma With Mesothelial Glandular Structures: A Diagnostic Pitfall.

Dedifferentiated liposarcoma shows a wide morphological spectrum. We present a case of dedifferentiated liposarcoma of the spermatic cord in a 66-year-old male that was initially misinterpreted as pseudosarcomatous proliferative funiculitis with mesothelial proliferation.

Pseudomyxoma Peritonei as a First Manifestation of KRAS-Mutated Urachal Mucinous Cystadenocarcinoma of the Bladder: A Case Report.

Only 28 cases of pseudomyxoma peritonei (PMP) arising from urachal neoplasms have been reported. We report one example of this extremely rare disease with KRAS mutational status in its spectrum of pathology. A 45-year-old woman presented with urachal frankly invasive mucinous cystadenocarcinoma confined to the dome of the bladder, which clinically manifested as PMP and was not detected at the first surgery. The primary tumour was revealed 6 months later because of its recurrence as PMP. Microscopic investigation revealed tubular adenoma and cystadenocarcinoma communicating with the bladder lumen and transitioning from the urachal urothelium to the mucinous epithelium. A urachal remnant was identified near the neoplasm. On immunohistochemistry, the tumour proved positive for CK7, CK20, CEA, and CDX2. Staining for ß-catenin revealed expression in both the cytoplasm and cell membrane. Mismatch repair protein expression was normal. Somatic KRAS-mutation (G12V) was revealed in tubular adenoma, cystadenocarcinoma, and mucinous carcinoma peritonei and may play an oncogenic role in the malignant transformation of urachal mucosa and the development of PMP.