[Use of hyperbaric oxygenation in various complications of craniocerebral injury in the acute period]. / Primenenie giperbaricheskoi oksigenatsii pri nekotorykh oslozhneniiakh cherepno-mozgovoi travmy v ostrom periode.

As the result of examination of 92 patients with acute craniocerebral trauma it was established that the use of HBO in the complex of therapeutic measures is permissible after removal of an intracranial hematoma despite occasional convulsive paroxysms, if there is no history of epilepsy. Exposure to HBO does not intensify or cause a recurrence of bleeding in traumatic subarachnoid hemorrhage and after operation for intracranial hematomas and brain contusion. Exposure to HBO does not cause an effect on the intensity of liquorrhea in the patients and does not lead to pneumocephalus or infection of the subarachnoid space.

[Pneumoencephalographic picture of changes in the cerebrospinal fluid containing spaces of patients who have suffered severe craniocerebral injuries]. / Pnevmoentsefalograficheskoe otobrazhenie izmenenii likvorosoderzhashchikh prostranstv u bol'nykh, perenesshikh tiazheluiu cherepno-mozgovuiu travmu.

Dynamic X-ray follow-ups were conducted after pneumo-encephalography (PEG) of 28 patients who had suffered a severe craniocerebral trauma (intracranial hematoma, crushing of the brain), and those suffering from a post-traumatic disease of the brain. PEG was carried out in periods from 3 months to 3 years after the trauma had occurred. X-ray control was established immediately after the endolumbar infusion of air into the intracranial cerebrospinal fluid spaces, and subsequently every 24 hours till the complete disappearance of air from the ventricles of the brain. Investigation showed that in most cases a more reliable picture of the size of the ventricles can be obtained not on the day that PEG is performed, but in remoter periods (24--48 hours). The internal hydrocephaly that occurs in most patients is evidently of aresorptive character. The expansion of subarachnoidal spaces, rather than their obliteration, is more characteristic of a traumatic disease of the brain, including post-traumatic epilepsy. These changes, however, are not pathognomonic for a complicated severe craniocerebral trauma.

[Brain stem lesions in acute cranio-cerebral injury]. / Porazheniia stvola golovnogo mozga pri ostroi cherepno-mozgovoi travme.

The authors analyzed the results of clinical and angiographical studies of 120 patients with acute severe brain traumas. In 76 cases brain stem lesions were diagnosed during life and in 22 cases diagnosed as a primary lesion. The postmortem data confirmed primary lesion of the stem structures only in 11 cases. The conclusion made by clinicians of the primary stem lesion was based mainly on an impetuously developing picture of stem disturbances during the first hours following trauma. The angiographical data demonstrated that each type of stem lesion in an acute brain trauma has a certain angiographic symptomatology. For a more correct diagnosis the authors recommend to perform cerebral angiography even in a typical clinical picture of primary stem lesion.