RESUMO
The authors give an account of the results of suture and decompression of the n. laryngeus recurrence in 15 patients with a mean follow up period of 17,6 months. A total of 12 nerves were sutured, one of them by means of an autograft from the n. suralis. A total of 11 nerves were decompressed. One patients after suture of the nerve and contralateral decompression did not attend a check-up examination. Of 11 sutured and checked nerves the results were very good in ten instances when a remarkable improvement of the voice was recorded as well as of the adduction movement and phonation closure of the vocal cords. In one instance the suture of the nerve failed. After decompression of the nerve in seven instances recovery of the function of the affected vocal cord occurred. After decompression of another two nerves the adduction movement of the vocal cord was restored, the voice became normal and phonation closure occurred. In one instance the nerve decompression failed. EMG examinations were made in three instances after suture of the nerve and in one instance after decompression of the nerve. The EMG examination correlated with the clinical finding. With regard to these results it is possible to recommend after injuries of the n. laryngeus recurrence its revision. The results of suture of the nerve or its decompression within five days after injury are very satisfactory in the group of patients treated by the authors.
Assuntos
Traumatismos do Nervo Laríngeo Recorrente , Nervo Laríngeo Recorrente/cirurgia , Adolescente , Adulto , Idoso , Feminino , Humanos , Complicações Intraoperatórias , Masculino , Pessoa de Meia-Idade , Suturas , Glândula Tireoide/cirurgia , Ferimentos e Lesões/etiologia , Ferimentos e Lesões/cirurgiaRESUMO
The author investigated the subclinical tremor on the extremity muscles in stutterers at rest, without speaking. This tremor occurred in the native EMG record from the musculus abductor digiti quinti during isometric contraction. Because it occurred in a muscle not involved in speaking and moreover at rest, without speaking, the author excludes the possibility that it could be an increase in the amplitude of physiological tremor; she concludes that the cause of this tremor are changes in the function of the structures responsible for motor feedback, especially in the extrapyramidal and cerebellar field.