RESUMO
While resource bootstrapping is widely employed by novice entrepreneurs,and social identities and decision logic are studied as important antecedents of entrepreneurs' adoption of resource bootstrapping behaviors, both scholars and practitioners have but a hazy understanding on the configuration effect of entrepreneur's social identities and decision logic on resource bootstrapping behaviors. In this paper, we combined the necessary condition analysis (NCA) and fuzzy set qualitative comparative analysis (fsQCA) to explore the configuration effects of entrepreneur's social identity and decision logic on resource bootstrapping behaviors. Analysis of 411 questionnaire date acquired from novice entrepreneurs, we found that none of single antecedent condition of both the entrepreneur's social identity and decision logic constitute the necessary condition of novice entrepreneurs' high resource bootstrapping behaviors. We also found a substitutive relationship between Darwinian social identity and communitary social identity that will lead the novice entrepreneurs with high dual effectual-causal decision logic to choose high joint utilization bootstrapping, and a complementary relationship between the three social identities of novice entrepreneurs such that those with high dual effectual-causal decision logic prefer different resource bootstrapping behaviors because of different social identities.Furthermore, we found that the core and edge conditions in the decision logic play different roles in the resource bootstrapping behaviors of novice entrepreneurs, with the core decision logic conditions having a greater impact on the resource bootstrapping behaviors. These findings deepen our understanding of the causal complexity among entrepreneur's social identity, decision logic and resource bootstrapping behaviors. It also provides theoretical guidance for effective resource integration for novice entrepreneurs facing resource constraints.
RESUMO
OBJECTIVE: To explore the role of aquaporin 1 (AQP1) in the initiation and development of hypertensive disorder complicating pregnancy (HDCP), and to analyze the relationship between AQP1 expression and ascites formation of patients with eclampsia. METHODS: Sixty inpatients with HDCP were recruited in the study, including 20 patients with gestational hypertension, 20 with mild preeclampsia, and 20 with severe preeclampsia. And 20 healthy pregnant women were taken as control. Immunohistochemistry was used to analyze AQP1 expressions in placenta, embryolemma and peritoneum, and B-mode ultrasonography was used to detect the ascites level of the patients. RESULTS: (1) AQP1 expression was detected in placenta, embryolemma and peritoneum. AQP1 was mainly located in endotheliocytes of blood vessels and blood capillaries in placenta, endothelial cells of amniotic membrane in embryolemma, and endotheliocytes of blood capillary and small veins in peritoneum. (2) The ascites incidence of HDCP patients (63%, 38/60) was higher than that of controls (10%, 2/20; P < 0.01). (3) The positive expressive rate of AQP1 in placenta of patients with HDCP (85%) was higher than that of controls (70%, P < 0.01). Furthermore, the AQP1 positive expressive rate from severe preeclampsia (90%) was obviously higher than that from gestational hypertension patients (80%, P < 0.05). (4) The AQP1 positive expressive rate in embryolemma from HDCP patients (87%) was lower than that of controls (95%, P < 0.05). The expressive rate from severe preeclampsia patients (80%) was obviously lower than that from gestational hypertension patients (95%, P < 0.05) and that of controls. (5) The AQP1 expressive rate in peritoneum from HDPC patients (82%) was higher than that of controls (70%, P < 0.01). The expressive rate of AQP1 from severe preeclampsia patients (90%) was obviously higher than that from gestational hypertension patients (75%, P < 0.05) and that of controls. CONCLUSIONS: The expression level of AQP1 of patients with preeclampsia increases in placenta and peritoneum and decreases in embryolemma, and holds correlation with the degree of HDCP. All these suggest that the changes in AQP1 expression may play an important role in the initiation and development of HDCP and may be one of the mechanisms for ascites formation.